Hae Jeong Youn, Ki Bbeum Kim, H. Han, I. An, K. Ahn
{"title":"23‐羟基甲酸通过减弱UVA诱导的氧化应激来保护人皮肤成纤维细胞","authors":"Hae Jeong Youn, Ki Bbeum Kim, H. Han, I. An, K. Ahn","doi":"10.1111/phpp.12294","DOIUrl":null,"url":null,"abstract":"Ultraviolet A (UVA), one of the major components of sunlight, can penetrate the dermal layer of the skin and generate reactive oxygen species (ROS). It causes alterations in the dermal connective tissue and gene expression, inflammation, photoaging, and DNA damage.","PeriodicalId":20060,"journal":{"name":"Photodermatology","volume":"C-34 1","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2017-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"9","resultStr":"{\"title\":\"23‐Hydroxytormentic acid protects human dermal fibroblasts by attenuating UVA‐induced oxidative stress\",\"authors\":\"Hae Jeong Youn, Ki Bbeum Kim, H. Han, I. An, K. Ahn\",\"doi\":\"10.1111/phpp.12294\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Ultraviolet A (UVA), one of the major components of sunlight, can penetrate the dermal layer of the skin and generate reactive oxygen species (ROS). It causes alterations in the dermal connective tissue and gene expression, inflammation, photoaging, and DNA damage.\",\"PeriodicalId\":20060,\"journal\":{\"name\":\"Photodermatology\",\"volume\":\"C-34 1\",\"pages\":\"\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2017-03-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"9\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Photodermatology\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.1111/phpp.12294\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Photodermatology","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1111/phpp.12294","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
23‐Hydroxytormentic acid protects human dermal fibroblasts by attenuating UVA‐induced oxidative stress
Ultraviolet A (UVA), one of the major components of sunlight, can penetrate the dermal layer of the skin and generate reactive oxygen species (ROS). It causes alterations in the dermal connective tissue and gene expression, inflammation, photoaging, and DNA damage.