姜黄素可防止运动引起的肌肉损伤小鼠的蛋白质氧化而不是脂质过氧化

Bambang Purwanto , Harjanto , I. Ketut Sudiana
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引用次数: 2

摘要

氧化应激被认为是运动引起肌肉损伤的重要机制。本研究旨在探讨姜黄素对运动诱导模型肌肉蛋白氧化和脂质过氧化的影响。以成年雄性健康小鼠为实验模型,分为姜黄素组、安慰剂组(仅玉米油组)和未治疗组。姜黄素治疗组蛋白羰基水平显著低于未治疗组(p =0.003)。相反,两组间丙二醛水平无显著差异(p = 0.092)。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Curcuminoid Prevents Protein Oxidation but not Lipid Peroxidation in Exercise Induced Muscle Damage Mouse

Oxidative stress is believed as underlined mechanism of exercise induced muscle damage. This study was aimed to investigate curcuminoid effect on protein oxidation and lipid peroxidation muscle of exercised induced model. Adult male healthy mice were used as experiment models, grouped in to curcuminoid treated, placebo (corn oil only treated) and untreated group. Protein carbonyl level was significantly lower in curcuminoid treated group compared with untreated group (p =0.003). In the contrary, the malondialdehyde level was not significantly different between those groups (p = 0.092).

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