{"title":"IIIa型发育不良的分子基础是什么?我们应该知道什么?为什么?","authors":"R. Andrade-Machado","doi":"10.4172/2472-0895.1000E112","DOIUrl":null,"url":null,"abstract":"Many papers are focused in the risk factors associated to temporal lobe epilepsy with HS. Complex febrile seizures, early head traumas, central nervous system infections or mild perinatal hypoxia have been associated with HS [3]. Although not totally elucidated, the molecular basis of HS is better known than the molecular basis of dysplasia type Ia or Ib. Thus, less is known about the complex mechanisms that provoke HS with dysplasia type Ia or Ib (dysplasia type IIIa).","PeriodicalId":81656,"journal":{"name":"Journal of epilepsy","volume":"2 1","pages":"1-3"},"PeriodicalIF":0.0000,"publicationDate":"2016-08-02","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.4172/2472-0895.1000E112","citationCount":"1","resultStr":"{\"title\":\"Which are the Molecular Basis of Dysplasia Type IIIa, What Should We Know and Why?\",\"authors\":\"R. Andrade-Machado\",\"doi\":\"10.4172/2472-0895.1000E112\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Many papers are focused in the risk factors associated to temporal lobe epilepsy with HS. Complex febrile seizures, early head traumas, central nervous system infections or mild perinatal hypoxia have been associated with HS [3]. Although not totally elucidated, the molecular basis of HS is better known than the molecular basis of dysplasia type Ia or Ib. Thus, less is known about the complex mechanisms that provoke HS with dysplasia type Ia or Ib (dysplasia type IIIa).\",\"PeriodicalId\":81656,\"journal\":{\"name\":\"Journal of epilepsy\",\"volume\":\"2 1\",\"pages\":\"1-3\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2016-08-02\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://sci-hub-pdf.com/10.4172/2472-0895.1000E112\",\"citationCount\":\"1\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of epilepsy\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.4172/2472-0895.1000E112\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of epilepsy","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.4172/2472-0895.1000E112","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Which are the Molecular Basis of Dysplasia Type IIIa, What Should We Know and Why?
Many papers are focused in the risk factors associated to temporal lobe epilepsy with HS. Complex febrile seizures, early head traumas, central nervous system infections or mild perinatal hypoxia have been associated with HS [3]. Although not totally elucidated, the molecular basis of HS is better known than the molecular basis of dysplasia type Ia or Ib. Thus, less is known about the complex mechanisms that provoke HS with dysplasia type Ia or Ib (dysplasia type IIIa).