更正“胰岛素样生长因子2 (IGF-2)增强bmp -9诱导的成骨分化和骨形成”

IF 5.1 1区医学 Q1 ENDOCRINOLOGY & METABOLISM

Journal of Bone and Mineral Research Pub Date : 2023-05-29 DOI:10.1002/jbmr.4830

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引用次数: 0

摘要

陈，姜丽，吴，黄，杰，贺，左，桂伟，张，伟，罗，强，施，强，张宝强，Wagner, ER，罗，杰，唐，M, Wietholt, C，罗，X，毕，Y，苏，Y，刘，B, Kim, SH，贺，CJ，胡，Y，沈，J, Rastegar, F，黄，E，高，杨，高，周建中，Reid, RR, Luu, HH, Haydon, RC，贺，T-C，邓，Z-L(2010)胰岛素样生长因子2 (IGF-2)增强bmp -9诱导的成骨分化和骨形成。骨外科杂志，2010;25(11): 2447 - 2459。在发表的文章中，作者发现图1C中的图像组装错误，其中“C3H10T1/2 (Ad-IGFP)”组的图像与图3A的“BMP9 + RFP”组的图像错误地复制了。更正后的数字如下所示。如更正后的图1C所示，这个错误并不会对原文的结论产生不利影响。作者对该错误给期刊和科学界带来的任何不便表示歉意。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Correction to ‘Insulin-Like Growth Factor 2 (IGF-2) Potentiates BMP-9-Induced Osteogenic Differentiation and Bone Formation’

Chen, L Jiang, W, Huang, J, He, B-C, Zuo, G-W, Zhang, W, Luo, Q, Shi, Q, Zhang, B-Q, Wagner, ER, Luo, J, Tang, M, Wietholt, C, Luo, X, Bi, Y, Su, Y, Liu, B, Kim, SH, He, CJ, Hu, Y, Shen, J, Rastegar, F, Huang, E, Gao, Y, Gao, J-L, Zhou, J-Z, Reid, RR, Luu, HH, Haydon, RC, He, T-C, Deng, Z-L (2010) Insulin-like growth factor 2 (IGF-2) potentiates BMP-9-induced osteogenic differentiation and bone formation. J Bone Miner Res. 2010; 25(11): 2447–2459. doi: https://doi.org/10.1002/jbmr.133

In the published article, the authors found an image assembly error in Figure 1C, in which the image for “C3H10T1/2 (Ad-IGFP)” group was erroneously duplicated with the image from the “BMP9 + RFP” group of Figure 3A. The corrected figure is shown below. As shown in the corrected Figure 1C, this error does not adversely impact the conclusion of the original work. The authors apologize for any inconvenience caused by the error to the journal and scientific community.

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来源期刊

Journal of Bone and Mineral Research 医学-内分泌学与代谢

CiteScore

11.30

自引率

6.50%

发文量

257

审稿时长

2 months

期刊介绍： The Journal of Bone and Mineral Research (JBMR) publishes highly impactful original manuscripts, reviews, and special articles on basic, translational and clinical investigations relevant to the musculoskeletal system and mineral metabolism. Specifically, the journal is interested in original research on the biology and physiology of skeletal tissues, interdisciplinary research spanning the musculoskeletal and other systems, including but not limited to immunology, hematology, energy metabolism, cancer biology, and neurology, and systems biology topics using large scale “-omics” approaches. The journal welcomes clinical research on the pathophysiology, treatment and prevention of osteoporosis and fractures, as well as sarcopenia, disorders of bone and mineral metabolism, and rare or genetically determined bone diseases.