植物器官MSH1是一种置换环特异性核酸内切酶。

IF 3.9 2区 生物学 Q2 CELL BIOLOGY
Alejandro Peñafiel-Ayala, Antolin Peralta-Castro, Josue Mora-Garduño, Paola García-Medel, Angie G Zambrano-Pereira, Corina Díaz-Quezada, María Jazmín Abraham-Juárez, Claudia G Benítez-Cardoza, Daniel B Sloan, Luis G Brieba
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引用次数: 0

摘要

MSH1是一种组织器靶向蛋白,它阻碍植物组织器基因组中异位重组和突变的积累。MSH1还调节核DNA的表观遗传学状态,其缺失诱导多种表型反应。MSH1是DNA错配修复蛋白MutS家族的一员,但含有一个额外的GIY-YIG核酸酶结构域,将其与该家族的其他成员区分开来。MSH1是如何阻碍重组和提高器官DNA遗传的保真度的尚不清楚。在这里,我们通过重组表达和纯化拟南芥全长MSH1(AtMSH1)来阐明其酶活性。AtMSH1是一种金属酶,对置换环(D-环)显示出强的结合亲和力。AtMSH1的DNA结合能力存在于其MutS结构域中,而不存在于其GIY-YIG结构域中。在存在二价金属离子的情况下,无论是否存在错配,AtMSH1都选择性地在D环处执行多个切口,但不在包括Holliday连接或dsDNA的其他DNA结构处执行。AtMSH1分解D环的选择性支持了这种酶在防止短重复序列之间重组中的作用。我们的研究结果表明,植物细胞器已经进化出以MSH1的抗重组活性为中心的新的DNA修复途径。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Plant Organellar MSH1 Is a Displacement Loop-Specific Endonuclease.

MutS HOMOLOG 1 (MSH1) is an organellar-targeted protein that obstructs ectopic recombination and the accumulation of mutations in plant organellar genomes. MSH1 also modulates the epigenetic status of nuclear DNA, and its absence induces a variety of phenotypic responses. MSH1 is a member of the MutS family of DNA mismatch repair proteins but harbors an additional GIY-YIG nuclease domain that distinguishes it from the rest of this family. How MSH1 hampers recombination and promotes fidelity in organellar DNA inheritance is unknown. Here, we elucidate its enzymatic activities by recombinantly expressing and purifying full-length MSH1 from Arabidopsis thaliana (AtMSH1). AtMSH1 is a metalloenzyme that shows a strong binding affinity for displacement loops (D-loops). The DNA-binding abilities of AtMSH1 reside in its MutS domain and not in its GIY-YIG domain, which is the ancillary nickase of AtMSH1. In the presence of divalent metal ions, AtMSH1 selectively executes multiple incisions at D-loops, but not other DNA structures including Holliday junctions or dsDNA, regardless of the presence or absence of mismatches. The selectivity of AtMSH1 to dismantle D-loops supports the role of this enzyme in preventing recombination between short repeats. Our results suggest that plant organelles have evolved novel DNA repair routes centered around the anti-recombinogenic activity of MSH1.

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来源期刊
Plant and Cell Physiology
Plant and Cell Physiology 生物-细胞生物学
CiteScore
8.40
自引率
4.10%
发文量
166
审稿时长
1.7 months
期刊介绍: Plant & Cell Physiology (PCP) was established in 1959 and is the official journal of the Japanese Society of Plant Physiologists (JSPP). The title reflects the journal''s original interest and scope to encompass research not just at the whole-organism level but also at the cellular and subcellular levels. Amongst the broad range of topics covered by this international journal, readers will find the very best original research on plant physiology, biochemistry, cell biology, molecular genetics, epigenetics, biotechnology, bioinformatics and –omics; as well as how plants respond to and interact with their environment (abiotic and biotic factors), and the biology of photosynthetic microorganisms.
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