铜酮诱导的小鼠脑脱髓鞘不是由于铜的耗尽。

IF 3.9 4区医学 Q2 NEUROSCIENCES

ASN NEURO Pub Date : 2022-01-01 DOI:10.1177/17590914221126367

Megan L Morgan, Wulin Teo, Yda Hernandez, Craig Brideau, Karen Cummins, Hedwich F Kuipers, Peter K Stys

引用次数: 3

摘要

总结性声明:CPZ的脱髓鞘作用不是由于铜缺乏，而是与CPZ + Cu复合物的急性毒性一致。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

Cuprizone-induced Demyelination in Mouse Brain is not due to Depletion of Copper.

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Cuprizone-induced Demyelination in Mouse Brain is not due to Depletion of Copper.

Summary statement: The demyelinating effects of CPZ are not due to Cu deficiency but are instead consistent with acute toxicity of a CPZ + Cu complex.

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来源期刊

ASN NEURO NEUROSCIENCES-

CiteScore

7.70

自引率

4.30%

发文量

审稿时长

>12 weeks

期刊介绍： ASN NEURO is an open access, peer-reviewed journal uniquely positioned to provide investigators with the most recent advances across the breadth of the cellular and molecular neurosciences. The official journal of the American Society for Neurochemistry, ASN NEURO is dedicated to the promotion, support, and facilitation of communication among cellular and molecular neuroscientists of all specializations.