急性锰暴露通过PI3K/Akt信号通路改变神经胶质细胞的翻译机制。

IF 3.9 4区医学 Q2 NEUROSCIENCES

ASN NEURO Pub Date : 2022-01-01 DOI:10.1177/17590914221131452

Jzmín Soto-Verdugo, Janisse Siva-Parra, Luisa C Hernández-Kelly, Arturo Ortega

引用次数: 0

摘要

摘要声明:我们在此证明，放射状胶质细胞短期暴露于锰(一种神经毒性金属)会诱导蛋白质合成，改变这些细胞的蛋白质库。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

Acute Manganese Exposure Modifies the Translation Machinery <i>via</i> PI3K/Akt Signaling in Glial Cells.

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Acute Manganese Exposure Modifies the Translation Machinery via PI3K/Akt Signaling in Glial Cells.

Summary statement: We demonstrate herein that short-term exposure of radial glia cells to Manganese, a neurotoxic metal, induces an effect on protein synthesis, altering the protein repertoire of these cells.

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来源期刊

ASN NEURO NEUROSCIENCES-

CiteScore

7.70

自引率

4.30%

发文量

审稿时长

>12 weeks

期刊介绍： ASN NEURO is an open access, peer-reviewed journal uniquely positioned to provide investigators with the most recent advances across the breadth of the cellular and molecular neurosciences. The official journal of the American Society for Neurochemistry, ASN NEURO is dedicated to the promotion, support, and facilitation of communication among cellular and molecular neuroscientists of all specializations.