产科硬膜外镇痛后硬膜外出血

K. Bouattour, G. Moyano-Tidou, A. Le Gouez, S. Martel-Jacob, F.-J. Mercier
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引用次数: 3

摘要

麻醉师经常站在第一线处理产后神经功能障碍,尽管硬膜外镇痛很少负责这些并发症。在一个34岁的产妇进行硬膜外镇痛以减轻自然分娩时的疼痛。由于胎儿心率异常,随后需要紧急剖腹产。拔管12小时后,产妇出现严重的右腿运动和感觉神经功能缺损,主要表现在L5和S1神经根,经神经科医生诊断为中枢神经根损伤。腰椎MRI发现L5椎体前部非压缩性硬膜外出血。分娩后硬膜外出血是一种罕见的并发症,可能危及功能预后。在某些情况下,可能很难区分由于分娩和分娩引起的上神经丛损伤与中央硬膜外镇痛相关的神经根病变。胎儿头部在骨盆边缘的压迫可能导致几个分化良好的神经区域的神经功能缺损,从而模仿麻醉诱导的髓周神经根病。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Saignement péridural suite à une analgésie péridurale obstétricale

Anaesthetists often stand in the front line to manage postpartum neurological deficits, although epidural analgesia is rarely responsible for these complications. An epidural analgesia was performed to relieve pain during spontaneous labor in a 34-year-old parturient. An emergency C-section was subsequently required due to fetal heart rate abnormalities. Twelve hours after catheter removal, the parturient developed a severe right leg motor and sensory neurological deficit, predominant on L5 and S1 roots and diagnosed by a neurologist as a central nerve root injury. Lumbar MRI identified a non-compressive epidural bleeding in front of the L5 vertebral body. Epidural bleeding after labor epidural analgesia is a rare complication that may jeopardize the functional prognosis. It may be difficult in some cases to differentiate an upper plexus injury due to labor and delivery from a central epidural analgesia-related nerve root lesion. Fetal head compression at the pelvic brim may induce neurological deficits in several well-differentiated nervous territories, thus mimicking an anaesthetic-induced perimedullar radiculopathy.

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