神经元网络的自适应和疲劳模型及非线性碎片方程的大时间渐近性。

IF 2.3 4区 医学 Q1 Neuroscience
Journal of Mathematical Neuroscience Pub Date : 2014-07-24 eCollection Date: 2014-01-01 DOI:10.1186/2190-8567-4-14
Khashayar Pakdaman, Benoît Perthame, Delphine Salort
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引用次数: 53

摘要

在具有适应和疲劳的神经网络模型的激励下,我们研究了一个保守的碎片化方程,该方程描述了在最后一次放电后经过时间为s的神经元的密度概率。在线性环境下,我们推广了laurenot和Perthame的论点,证明了指数衰减到稳态。这个扩展允许我们处理系数有很大的变化,而不是常数系数。在此论点的另一个扩展中,我们处理了一个弱非线性的情况,并证明了网络中的完全不同步。对于更大的非线性,我们采用两个“极端”情况,对碎片项对网络中神经元同步出现的影响进行了数值研究。数学学科分类(2000)2010:35B40, 35F20, 35R09, 92B20。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Adaptation and fatigue model for neuron networks and large time asymptotics in a nonlinear fragmentation equation.

Adaptation and fatigue model for neuron networks and large time asymptotics in a nonlinear fragmentation equation.

Adaptation and fatigue model for neuron networks and large time asymptotics in a nonlinear fragmentation equation.

Adaptation and fatigue model for neuron networks and large time asymptotics in a nonlinear fragmentation equation.

Motivated by a model for neural networks with adaptation and fatigue, we study a conservative fragmentation equation that describes the density probability of neurons with an elapsed time s after its last discharge. In the linear setting, we extend an argument by Laurençot and Perthame to prove exponential decay to the steady state. This extension allows us to handle coefficients that have a large variation rather than constant coefficients. In another extension of the argument, we treat a weakly nonlinear case and prove total desynchronization in the network. For greater nonlinearities, we present a numerical study of the impact of the fragmentation term on the appearance of synchronization of neurons in the network using two "extreme" cases. Mathematics Subject Classification (2000)2010: 35B40, 35F20, 35R09, 92B20.

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来源期刊
Journal of Mathematical Neuroscience
Journal of Mathematical Neuroscience Neuroscience-Neuroscience (miscellaneous)
自引率
0.00%
发文量
0
审稿时长
13 weeks
期刊介绍: The Journal of Mathematical Neuroscience (JMN) publishes research articles on the mathematical modeling and analysis of all areas of neuroscience, i.e., the study of the nervous system and its dysfunctions. The focus is on using mathematics as the primary tool for elucidating the fundamental mechanisms responsible for experimentally observed behaviours in neuroscience at all relevant scales, from the molecular world to that of cognition. The aim is to publish work that uses advanced mathematical techniques to illuminate these questions. It publishes full length original papers, rapid communications and review articles. Papers that combine theoretical results supported by convincing numerical experiments are especially encouraged. Papers that introduce and help develop those new pieces of mathematical theory which are likely to be relevant to future studies of the nervous system in general and the human brain in particular are also welcome.
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