{"title":"减重手术后β细胞功能的变化。","authors":"Adrian Vella","doi":"10.1152/physiol.00003.2014","DOIUrl":null,"url":null,"abstract":"The study of diabetes in humans has been hampered to some extent by the relative inaccessibility of a key organ in the pathogenesis of this disease: the islet of Langerhans and, more importantly, the insulin secretory machinery that are β-cells ([4][1], [16][2]). This has resulted in a relative","PeriodicalId":520753,"journal":{"name":"Physiology (Bethesda, Md.)","volume":" ","pages":"84-5"},"PeriodicalIF":10.3000,"publicationDate":"2014-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1152/physiol.00003.2014","citationCount":"3","resultStr":"{\"title\":\"β-cell function after weight-loss induced by bariatric surgery.\",\"authors\":\"Adrian Vella\",\"doi\":\"10.1152/physiol.00003.2014\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"The study of diabetes in humans has been hampered to some extent by the relative inaccessibility of a key organ in the pathogenesis of this disease: the islet of Langerhans and, more importantly, the insulin secretory machinery that are β-cells ([4][1], [16][2]). This has resulted in a relative\",\"PeriodicalId\":520753,\"journal\":{\"name\":\"Physiology (Bethesda, Md.)\",\"volume\":\" \",\"pages\":\"84-5\"},\"PeriodicalIF\":10.3000,\"publicationDate\":\"2014-03-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://sci-hub-pdf.com/10.1152/physiol.00003.2014\",\"citationCount\":\"3\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Physiology (Bethesda, Md.)\",\"FirstCategoryId\":\"3\",\"ListUrlMain\":\"https://doi.org/10.1152/physiol.00003.2014\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Physiology (Bethesda, Md.)","FirstCategoryId":"3","ListUrlMain":"https://doi.org/10.1152/physiol.00003.2014","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
β-cell function after weight-loss induced by bariatric surgery.
The study of diabetes in humans has been hampered to some extent by the relative inaccessibility of a key organ in the pathogenesis of this disease: the islet of Langerhans and, more importantly, the insulin secretory machinery that are β-cells ([4][1], [16][2]). This has resulted in a relative
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