铁下垂:一种非凋亡细胞死亡的铁依赖性形式。

IF 45.5 1区 生物学 Q1 BIOCHEMISTRY & MOLECULAR BIOLOGY
Cell Pub Date : 2012-05-25 DOI:10.1016/j.cell.2012.03.042
Scott J Dixon, Kathryn M Lemberg, Michael R Lamprecht, Rachid Skouta, Eleina M Zaitsev, Caroline E Gleason, Darpan N Patel, Andras J Bauer, Alexandra M Cantley, Wan Seok Yang, Barclay Morrison, Brent R Stockwell
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引用次数: 7629

摘要

非凋亡形式的细胞死亡可能有助于选择性消除某些肿瘤细胞或在特定病理状态下被激活。致癌性ras选择性致死性小分子擦除素触发一种独特的铁依赖性非凋亡细胞死亡形式,我们称之为铁凋亡。铁死亡依赖于细胞内的铁,而不是其他金属,并且在形态、生化和遗传上不同于细胞凋亡、坏死和自噬。我们在器官型大鼠脑切片中发现了小分子铁他汀-1作为癌细胞铁凋亡和谷氨酸诱导的细胞死亡的有效抑制剂,这表明这两个过程之间存在相似性。事实上,像谷氨酸一样,erastin抑制胱氨酸/谷氨酸反转运体(系统x(c)(-))对胱氨酸的摄取,在细胞的抗氧化防御中产生空洞,最终导致铁依赖性氧化性死亡。因此,铁下垂的激活导致某些癌细胞的非凋亡性破坏,而抑制这一过程可能保护生物体免受神经变性。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Ferroptosis: an iron-dependent form of nonapoptotic cell death.

Nonapoptotic forms of cell death may facilitate the selective elimination of some tumor cells or be activated in specific pathological states. The oncogenic RAS-selective lethal small molecule erastin triggers a unique iron-dependent form of nonapoptotic cell death that we term ferroptosis. Ferroptosis is dependent upon intracellular iron, but not other metals, and is morphologically, biochemically, and genetically distinct from apoptosis, necrosis, and autophagy. We identify the small molecule ferrostatin-1 as a potent inhibitor of ferroptosis in cancer cells and glutamate-induced cell death in organotypic rat brain slices, suggesting similarities between these two processes. Indeed, erastin, like glutamate, inhibits cystine uptake by the cystine/glutamate antiporter (system x(c)(-)), creating a void in the antioxidant defenses of the cell and ultimately leading to iron-dependent, oxidative death. Thus, activation of ferroptosis results in the nonapoptotic destruction of certain cancer cells, whereas inhibition of this process may protect organisms from neurodegeneration.

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来源期刊
Cell
Cell 生物-生化与分子生物学
CiteScore
110.00
自引率
0.80%
发文量
396
审稿时长
2 months
期刊介绍: Cells is an international, peer-reviewed, open access journal that focuses on cell biology, molecular biology, and biophysics. It is affiliated with several societies, including the Spanish Society for Biochemistry and Molecular Biology (SEBBM), Nordic Autophagy Society (NAS), Spanish Society of Hematology and Hemotherapy (SEHH), and Society for Regenerative Medicine (Russian Federation) (RPO). The journal publishes research findings of significant importance in various areas of experimental biology, such as cell biology, molecular biology, neuroscience, immunology, virology, microbiology, cancer, human genetics, systems biology, signaling, and disease mechanisms and therapeutics. The primary criterion for considering papers is whether the results contribute to significant conceptual advances or raise thought-provoking questions and hypotheses related to interesting and important biological inquiries. In addition to primary research articles presented in four formats, Cells also features review and opinion articles in its "leading edge" section, discussing recent research advancements and topics of interest to its wide readership.
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