杏仁核和海马体中LTP和ncam的神经调节剂在应激反应中的作用。

EXS Pub Date : 2006-01-01 DOI:10.1007/978-3-7643-7772-4_7

Adi Guterman, Gal Richter-Levin

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引用次数: 9

摘要

可能，在情绪事件开始时，应激激素允许调节可塑性。具体来说，CORT和NE应激激素参与杏仁核和海马体记忆巩固过程的调节。此外，与肾上腺素受体结合的糖皮质激素和去甲肾上腺素也参与了杏仁核和海马中NCAM多唾液化的调节。在特别具有挑战性的环境下，与psa相关的突触重塑被动员起来形成记忆。

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Neuromodulators of LTP and NCAMs in the amygdala and hippocampus in response to stress.

Possibly, at the onset of an emotional event the stress hormones permissively mediate plasticity. Specifically, CORT and NE stress hormones participate in modulation of memory consolidation processes in both the amygdala and the hippocampus. In addition, glucocorticoids and norepinephrin bound to adrenoceptors are also involved in modulating the regulation of NCAM polysialylation both in the amygdala and in the hippocampus. PSA-related synaptic remodeling is mobilized for memory formation in particularly challenging circumstances.

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