实验性急性胰腺炎心脏超微结构。

A Saulea, S Costin, V Rotari
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引用次数: 0

摘要

心肌超微结构改变引起的大鼠再现急性胰腺炎:微循环血管结构的影响,导致间质水肿和心肌细胞缺氧。2. 细胞外观粗糙,肌原纤维过度收缩,可能是由于肌膜结构缺陷引起的钙过量充盈所致。3.心肌细胞的细胞内水肿和它们的钙充盈可能是由肉瘤结构的缺陷引起的。4. 在最近的时期,心肌细胞和结缔组织对病变的反应表现为心肌细胞代偿性肥大和心肌基质的胶原化。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Heart ultrastructure in experimental acute pancreatitis.

Acute pancreatitis reproduced on white rats provoked following modifications of myocardium ultrastructure: 1. Affection of the microcirculation vessels structure, what induce interstitial edema and cardiomyocytes hypoxia. 2. The cells appearance of a rough type with the overcontracted myofibrils making up, probably occurs as a result of supercharging with calcium can be caused by deficiencies in the sarcolemma structure. 3. The intracellular edema of the cardiomyocytes and their supercharging with calcium can be caused by deficiencies in the sarcoma structure. 4. In the latest periods, the cardiomyocytes and the connective tissue reactions to the lesion is represented of compensatory hypertrophy of the cardiomyocytes and collagenizing the myocardium stroma.

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