{"title":"假设","authors":"D.F. Davies","doi":"10.1016/S0368-1319(69)80013-X","DOIUrl":null,"url":null,"abstract":"<div><p>Evidence is presented for an immunological disorder in atherogenesis. At present milk protein is the only antigen known to be involved but there may be others. This disorder offers a means of integrating many existing facts about atherosclerosis that are otherwise apparently unrelated. The hypothesis embraces both the filtration concept and also the thrombogenic theory.</p></div>","PeriodicalId":78351,"journal":{"name":"Journal of atherosclerosis research","volume":"10 2","pages":"Pages 253-259"},"PeriodicalIF":0.0000,"publicationDate":"1969-09-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/S0368-1319(69)80013-X","citationCount":"41","resultStr":"{\"title\":\"Hypothesis\",\"authors\":\"D.F. Davies\",\"doi\":\"10.1016/S0368-1319(69)80013-X\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<div><p>Evidence is presented for an immunological disorder in atherogenesis. At present milk protein is the only antigen known to be involved but there may be others. This disorder offers a means of integrating many existing facts about atherosclerosis that are otherwise apparently unrelated. The hypothesis embraces both the filtration concept and also the thrombogenic theory.</p></div>\",\"PeriodicalId\":78351,\"journal\":{\"name\":\"Journal of atherosclerosis research\",\"volume\":\"10 2\",\"pages\":\"Pages 253-259\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1969-09-10\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://sci-hub-pdf.com/10.1016/S0368-1319(69)80013-X\",\"citationCount\":\"41\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of atherosclerosis research\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://www.sciencedirect.com/science/article/pii/S036813196980013X\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of atherosclerosis research","FirstCategoryId":"1085","ListUrlMain":"https://www.sciencedirect.com/science/article/pii/S036813196980013X","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Evidence is presented for an immunological disorder in atherogenesis. At present milk protein is the only antigen known to be involved but there may be others. This disorder offers a means of integrating many existing facts about atherosclerosis that are otherwise apparently unrelated. The hypothesis embraces both the filtration concept and also the thrombogenic theory.
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