Nikole Rautiainen, Eveliina Brandt, Kaisa Kettunen, Outi Elomaa, Sirpa Kivirikko, Leila Jeskanen, Katriina Lappalainen, Nelli Sjöblom, Juha Kere, Katariina Hannula-Jouppi, Liisa Harjama
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引用次数: 0
摘要
Costello综合征(CS)是一种罕见的显性HRAS RASopathy,以卷发、心脏异常、颅面异常和发育迟缓为特征。HRAS密码子58、59和60变异与较轻的表型相关。我们描述了一个三代家族,该家族具有以前未报道的杂合HRAS变异c.175G> a (p.Ala59Thr),导致主要的外胚层表型。采用外显子组和Sanger测序进行遗传分析。进行皮肤和心脏评估,包括皮肤活检和头发样本显微镜检查。一名14岁的先证女、她的双胞胎姐妹、母亲、母亲的父亲和母亲的同父异母兄弟都有一个共同的表型,即头发毛茸茸、稀疏、睫毛卷曲、眉毛稀疏、发性红斑、毛糙角化病、掌跖角化病和耳朵后旋低。一名患者在出生后需要进行胃造口术,但没有其他典型的CS特征,包括颅面异常、肥厚性心肌病和智力残疾。我们进行了一项支持与HRAS密码子58-60变异相关的减弱CS表型的比较。总之,HRAS c.175G>A (p.Ala59Thr)主要引起外胚层表型,与与经典CS不同的密码子58-60相关的较轻的HRAS相关ras病变一致。对于具有外胚层和cs样特征的患者,应考虑HRAS变异,以便进行准确的遗传诊断和有针对性的治疗。
The HRAS Variant c.175G>A (p.Ala59Thr) Causes a Predominantly Ectodermal Phenotype Lacking Classic Costello Syndrome Features.
Costello syndrome (CS) is a rare dominant HRAS RASopathy characterized by curly hair, cardiac abnormalities, craniofacial anomalies, and developmental delay. HRAS codon 58, 59, and 60 variants are associated with milder phenotypes. We describe a three-generation family with a previously unreported heterozygous HRAS variant c.175G>A (p.Ala59Thr) causing a predominantly ectodermal phenotype. Exome and Sanger sequencing were used for genetic analysis. Dermatological and cardiac evaluations were performed, including skin biopsy and hair sample microscopy. A 14-year-old proband, her twin sister, mother, mother's father, and mother's paternal half-brother all shared a phenotype of woolly and sparse hair, curly eyelashes, sparse eyebrows, ulerythema ophryogenes, keratosis pilaris, palmoplantar keratoderma, and low-set posteriorly rotated ears. One patient required a gastrostomy after birth but otherwise classic CS features, including craniofacial anomalies, hypertrophic cardiomyopathy, and intellectual disability, were absent. We conducted a comparison supporting the attenuated CS phenotype associated with HRAS codon 58-60 variants. In conclusion, HRAS c.175G>A (p.Ala59Thr) causes predominantly an ectodermal phenotype, consistent with milder HRAS-related RASopathies involving codons 58-60 distinguishable from classic CS. HRAS variants should be considered in patients with ectodermal and CS-like features for accurate genetic diagnosis and targeted management.
期刊介绍:
The American Journal of Medical Genetics - Part A (AJMG) gives you continuous coverage of all biological and medical aspects of genetic disorders and birth defects, as well as in-depth documentation of phenotype analysis within the current context of genotype/phenotype correlations. In addition to Part A , AJMG also publishes two other parts:
Part B: Neuropsychiatric Genetics , covering experimental and clinical investigations of the genetic mechanisms underlying neurologic and psychiatric disorders.
Part C: Seminars in Medical Genetics , guest-edited collections of thematic reviews of topical interest to the readership of AJMG .