更正：RAS/MEK/ERK信号的激活驱动原发性肝癌的胆道分化

IF 25.8 1区医学 Q1 GASTROENTEROLOGY & HEPATOLOGY

Gut Pub Date : 2025-11-01 DOI:10.1136/gutjnl-2024-333238corr1

BMJ Publishing Group Ltd and British Society of Gastroenterology

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引用次数: 0

摘要

Rösner T, Rupp C, Lechler C，等。RAS/MEK/ERK信号的激活驱动原发性肝癌的胆道分化肠道74:1653 2025;1666年。在已发表的文章中，图2-4没有按正确的顺序出现。图2 RPK肝肿瘤由肝细胞（HEP）发展而来。(A)以HEP（红色）或胆管细胞（绿色）为靶点注射AAV-TBG-Cre或tam后，雄性RPK小鼠的存活率。

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Correction: Activation of RAS/MEK/ERK signalling drives biliary differentiation in primary liver cancer

Rösner T, Rupp C, Lechler C, et al . Activation of RAS/MEK/ERK signalling drives biliary differentiation in primary liver cancer. Gut 2025;74:1653-1666. In the published article, figures 2–4 do not appear in the correct order. The correct figures are below: Figure 2 RPK liver tumours develop from hepatocytes (HEP). (A) Survival of male RPK mice after injection of AAV-TBG-Cre or tam in RPK lines targeting HEP (red) or bile duct cells (green) in …

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来源期刊

Gut 医学-胃肠肝病学

CiteScore

45.70

自引率

2.40%

发文量

284

审稿时长

1.5 months

期刊介绍： Gut is a renowned international journal specializing in gastroenterology and hepatology, known for its high-quality clinical research covering the alimentary tract, liver, biliary tree, and pancreas. It offers authoritative and current coverage across all aspects of gastroenterology and hepatology, featuring articles on emerging disease mechanisms and innovative diagnostic and therapeutic approaches authored by leading experts. As the flagship journal of BMJ's gastroenterology portfolio, Gut is accompanied by two companion journals: Frontline Gastroenterology, focusing on education and practice-oriented papers, and BMJ Open Gastroenterology for open access original research.