在败血症中训练免疫:探索与长期心脏代谢紊乱的分子联系。

IF 3.1 4区 医学 Q3 IMMUNOLOGY
Rijhul Lahariya, Gargee Anand, Bandana Kumari
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引用次数: 0

摘要

脓毒症是一种危及生命的全身性感染,长期以来一直被认为具有直接风险,但其对健康的长期影响越来越明显,特别是在慢性心脏代谢疾病(cms)如动脉粥样硬化、胰岛素抵抗和血脂异常的易感幸存者中。这个过程的核心是训练免疫,其中先天免疫细胞如单核细胞、巨噬细胞和中性粒细胞在败血症后经历长期的表观遗传重编程。这种重编程由NF-κB、mTOR和脂质代谢改变等分子途径维持,导致慢性炎症、氧化应激和代谢功能障碍,导致长期心血管疾病(cvd)和败血症后代谢紊乱。这篇综述探讨了训练免疫连接败血症和CMDs的关键机制,特别关注表观遗传修饰,如组蛋白乙酰化、DNA甲基化和线粒体改变。我们讨论了训练免疫如何增强免疫细胞激活,导致持续的低度炎症,脂质失调和胰岛素敏感性受损,所有这些都使败血症幸存者易患心血管疾病。此外,我们强调针对训练免疫的潜在治疗方法,包括他汀类药物,可减少炎症和免疫重编程;二甲双胍,通过激活AMPK和减少氧化应激来恢复代谢平衡;富马酸二甲酯(DMF),一种有效的Nrf2激活剂,可以抵消炎症;益生菌有助于恢复肠道菌群平衡,限制内毒素引起的炎症。这些疗法为缓解长期代谢功能障碍和减少败血症后CMDs的发生率提供了有希望的策略。了解这些机制并制定有针对性的干预措施可能最终有助于预防败血症幸存者的慢性心血管和代谢性疾病,并改善长期预后。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Trained Immunity in sepsis: Exploring the molecular link to long-term cardiometabolic disorders.

Sepsis, a life-threatening systemic infection, has long been recognized for its immediate risks, but its long-term consequences on health are increasingly evident, particularly in predisposing survivors to chronic cardiometabolic disorders (CMDs) such as atherosclerosis, insulin resistance, and dyslipidemia. Central to this process is trained immunity, where innate immune cells like monocytes, macrophages, and neutrophils undergo long-lasting epigenetic reprogramming after sepsis. This reprogramming, sustained by molecular pathways such as NF-κB, mTOR, and altered lipid metabolism, drives chronic inflammation, oxidative stress, and metabolic dysfunction, contributing to long-term cardiovascular diseases (CVDs) and metabolic disorders post-sepsis. This review explores the key mechanisms through which trained immunity bridges sepsis and CMDs, particularly focusing on epigenetic modifications such as histone acetylation, DNA methylation, and mitochondrial alterations. We discuss how trained immunity enhances immune cell activation, leading to persistent low-grade inflammation, lipid dysregulation, and impaired insulin sensitivity, all of which predispose sepsis survivors to CVDs. Additionally, we highlight potential therapeutic approaches targeting trained immunity, including statins, which reduce inflammation and immune reprogramming; metformin, which restores metabolic balance by activating AMPK and reducing oxidative stress; dimethyl fumarate (DMF), a potent Nrf2 activator that counteracts inflammation; and probiotics, which help restore gut microbiota balance and limit endotoxin-driven inflammation. These therapies offer promising strategies to mitigate long-term metabolic dysfunction and reduce the incidence of CMDs following sepsis. Understanding these mechanisms and developing targeted interventions may ultimately help prevent chronic cardiovascular and metabolic diseases in sepsis survivors and improve long-term outcomes.

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来源期刊
Immunologic Research
Immunologic Research 医学-免疫学
CiteScore
6.90
自引率
0.00%
发文量
83
审稿时长
6-12 weeks
期刊介绍: IMMUNOLOGIC RESEARCH represents a unique medium for the presentation, interpretation, and clarification of complex scientific data. Information is presented in the form of interpretive synthesis reviews, original research articles, symposia, editorials, and theoretical essays. The scope of coverage extends to cellular immunology, immunogenetics, molecular and structural immunology, immunoregulation and autoimmunity, immunopathology, tumor immunology, host defense and microbial immunity, including viral immunology, immunohematology, mucosal immunity, complement, transplantation immunology, clinical immunology, neuroimmunology, immunoendocrinology, immunotoxicology, translational immunology, and history of immunology.
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