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引用次数: 0
摘要
引用本文:朱竑,郑军,周艳,吴涛,朱涛,“PRMT5通过rsad2介导的NF-κB信号参与原发性干燥综合征(pSS)患者B细胞过度激活”,《中华免疫杂志》,第11期,no. 11。12 (2023): e1102, https://doi.org/10.1002/iid3.1102。上述文章于2023年12月13日在线发表在Wiley在线图书馆(wileyonlinelibrary.com)上,经作者同意撤回;杂志主编Marc Veldhoen;和约翰威利父子有限公司。由于在图4C和6E之间发现了p-p65 western blot条带的重复,因此同意撤回。这种复制是由同一条带的旋转版本组成的,它们被用来代表不同的科学条件。在参与者简介和供资资料中也注意到差异。该研究包括了异常高比例的男性参与者,他们的疾病已知主要影响女性。此外,资助声明缺乏机构归属,似乎与研究的重点不一致。作者没有回应就这些担忧发表评论或提供支持数据的邀请。因此,编辑们认为结果和结论受到了损害。
RETRACTION: PRMT5 Participates in B Cell Overactivation in Patients With Primary Sjogren's Syndrome (pSS) Through RSAD2-Mediated NF-κB Signaling
RETRACTION: H. Zhu, J. Zheng, Y. Zhou, T. Wu, and T. Zhu, “PRMT5 Participates in B Cell Overactivation in Patients With Primary Sjogren's Syndrome (pSS) Through RSAD2-Mediated NF-κB Signaling,” Immunity, Inflammation and Disease 11, no. 12 (2023): e1102, https://doi.org/10.1002/iid3.1102.
The above article, published online on 13 December 2023 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the authors; the journal Editor-in-Chief, Marc Veldhoen; and John Wiley & Sons, Ltd. The retraction has been agreed upon due to the identification of duplication of the p-p65 western blot bands between Figures 4C and 6E. The duplication consists of rotated versions of the same bands, which were used to represent different scientific conditions. Discrepancies were also noted in the participant profile and funding information. The study included an unusually high proportion of male participants for a condition known to predominantly affect women. Additionally, the funding statement lacked institutional attribution and appeared misaligned with the study's focus. The authors did not respond to invitations to comment on the concerns or provide supporting data. As a result, the editors consider the results and conclusions to be compromised.
期刊介绍:
Immunity, Inflammation and Disease is a peer-reviewed, open access, interdisciplinary journal providing rapid publication of research across the broad field of immunology. Immunity, Inflammation and Disease gives rapid consideration to papers in all areas of clinical and basic research. The journal is indexed in Medline and the Science Citation Index Expanded (part of Web of Science), among others. It welcomes original work that enhances the understanding of immunology in areas including:
• cellular and molecular immunology
• clinical immunology
• allergy
• immunochemistry
• immunogenetics
• immune signalling
• immune development
• imaging
• mathematical modelling
• autoimmunity
• transplantation immunology
• cancer immunology