gcn2介导的综合应激反应可减轻牙周炎。

IF 5.9 1区 医学 Q1 DENTISTRY, ORAL SURGERY & MEDICINE
D Huang,Q Li,S Peng,Y Li,Q Yin,X Yang,X Gan,Y Wang,X Li,Y Zhao,Y Guo,W Lin,Y Li,N Feng,Q Yuan
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引用次数: 0

摘要

综合应激反应(ISR)是由一般控制非抑制2 (GCN2)调控的,对维持组织稳态至关重要,但其在牙周炎中的作用仍知之甚少。通过对患者牙龈活检和结扎诱导的小鼠牙周炎模型的转录组学分析和免疫组化,我们证明gcn2介导的ISR在人和小鼠牙周炎中都被激活,并且主要在巨噬细胞中起作用。使用Gcn2-/-小鼠,我们发现Gcn2缺失加剧了实验性牙周炎的牙龈炎症和骨质流失。在机制上,大量RNA-seq和体外实验显示GCN2的缺失会损害自噬,并导致NLRP3/CASPASE1炎性体通路的过度激活。值得注意的是,局部给药氟醌(一种GCN2激活剂)以GCN2依赖的方式减轻口腔炎症和组织破坏。总之,我们的工作强调了GCN2介导的ISR在口腔黏膜中的保护作用,并表明GCN2是治疗牙周炎的有希望的靶点。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
GCN2-Mediated Integrated Stress Response Attenuates Periodontitis.
The integrated stress response (ISR), regulated by general control nonderepressible 2 (GCN2), is essential for maintaining tissue homeostasis, yet its role in periodontitis remains poorly understood. Here, through transcriptomic analysis and immunohistochemistry of gingival biopsies from patients and a ligature-induced mouse periodontitis model, we demonstrate that GCN2-mediated ISR is activated in both human and mouse periodontitis and mainly functioned in macrophages. Using Gcn2-/- mice, we show that Gcn2 deletion exacerbates gingival inflammation and bone loss in experimental periodontitis. Mechanistically, bulk RNA-seq and in vitro assays revealed that the loss of GCN2 impairs autophagy and leads to overactivation of the NLRP3/CASPASE1 inflammasome pathway. Notably, local administration of halofuginone, a GCN2 activator, mitigates oral inflammation and tissue destruction in a GCN2-dependent manner. In summary, our work highlights the protective role of the GCN2-mediated ISR in oral mucosa and indicates GCN2 as a promising therapeutic target for periodontitis.
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来源期刊
Journal of Dental Research
Journal of Dental Research 医学-牙科与口腔外科
CiteScore
15.30
自引率
3.90%
发文量
155
审稿时长
3-8 weeks
期刊介绍: The Journal of Dental Research (JDR) is a peer-reviewed scientific journal committed to sharing new knowledge and information on all sciences related to dentistry and the oral cavity, covering health and disease. With monthly publications, JDR ensures timely communication of the latest research to the oral and dental community.
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