5-FU耐药性通过adam10介导的PD-L1脱落和肿瘤微环境重塑促进食管鳞状细胞癌的免疫逃避。

IF 5 3区 医学 Q2 IMMUNOLOGY
Immunology Pub Date : 2025-09-16 DOI:10.1111/imm.70038
Bangwu Cai, Xiaowen Feng, Shujuan Luo, Aididar Nurbahati, Hong Cui, Tianyuan Peng, Wei Wang, Huifang Li, Qing Liu, Xiaomei Lu, Shutao Zheng
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引用次数: 0

摘要

化疗耐药仍然是食管鳞状细胞癌(ESCC)治疗的主要挑战,特别是对5-氟尿嘧啶(5-FU)的耐药。这项研究揭示了5-FU耐药性是如何重新编程肿瘤微环境的,主要是通过上调ADAM10和释放可溶性PD-L1,共同促进免疫逃避。利用5-FU-AKR耐药小鼠ESCC细胞系(5-FU-AKR)及其亲代对应细胞系,我们应用第三代DNA测序、蛋白质组学分析和单细胞RNA测序来揭示与耐药相关的分子和细胞转移。我们发现ADAM10在5-FU-AKR细胞中显著上调,促进可溶性PD-L1的释放,从而限制CD8+ T细胞的浸润。异种移植模型进一步证明了在5- fu耐药肿瘤中增强的致瘤性和免疫排斥。这些发现强调了ADAM10驱动免疫抑制的新机制,表明靶向ADAM10- pd - l1轴可能恢复抗肿瘤免疫并改善5- fu耐药ESCC的治疗结果。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
5-FU Resistance Facilitates Immune Evasion in Esophageal Squamous Cell Carcinoma Through ADAM10-Mediated PD-L1 Shedding and Tumour Microenvironment Remodelling.

Chemoresistance remains a major challenge in esophageal squamous cell carcinoma (ESCC) therapy, particularly resistance to 5-Fluorouracil (5-FU). This study uncovers how 5-FU resistance reprograms the tumour microenvironment, primarily through the up-regulation of ADAM10 and the release of soluble PD-L1, which collectively facilitate immune evasion. Using a 5-FU-resistant AKR mouse ESCC cell line (5-FU-AKR) and its parental counterpart, we applied third-generation DNA sequencing, proteomic analysis, and single-cell RNA sequencing to unravel the resistance-associated molecular and cellular shifts. We found that ADAM10 is significantly up-regulated in 5-FU-AKR cells, promoting soluble PD-L1 release, thereby limiting CD8+ T cell infiltration. Xenograft models further demonstrated enhanced tumourigenicity and immune exclusion in 5-FU-resistant tumours. These findings highlight a novel mechanism of immune suppression driven by ADAM10, suggesting that targeting the ADAM10-PD-L1 axis may restore anti-tumour immunity and improve treatment outcomes for 5-FU-resistant ESCC.

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来源期刊
Immunology
Immunology 医学-免疫学
CiteScore
11.90
自引率
1.60%
发文量
175
审稿时长
4-8 weeks
期刊介绍: Immunology is one of the longest-established immunology journals and is recognised as one of the leading journals in its field. We have global representation in authors, editors and reviewers. Immunology publishes papers describing original findings in all areas of cellular and molecular immunology. High-quality original articles describing mechanistic insights into fundamental aspects of the immune system are welcome. Topics of interest to the journal include: immune cell development, cancer immunology, systems immunology/omics and informatics, inflammation, immunometabolism, immunology of infection, microbiota and immunity, mucosal immunology, and neuroimmunology. The journal also publishes commissioned review articles on subjects of topical interest to immunologists, and commissions in-depth review series: themed sets of review articles which take a 360° view of select topics at the heart of immunological research.
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