修正“针对β淀粉样蛋白和α-突触核蛋白的单一和联合免疫治疗方法在路易小体样痴呆模型中的作用”

IF 11.1 1区 医学 Q1 CLINICAL NEUROLOGY
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引用次数: 0

摘要

Mandler M, Rockenstein E, Overk C,等。针对β淀粉样蛋白和α-突触核蛋白的单一和联合免疫治疗对路易小体样痴呆模型的影响。老年痴呆症。2025;15(9):1133-1148。作者很遗憾,在图4G和图5D的组装过程中出现了一个错误,图4G用错误的面板来表示黑河鼠的APP/α-syn tg AD02和AD02/PD-AFF1条件,图5D用错误的面板来表示海马的APP/α-syn tg AD02/PD-AFF1。更正后的数字如下附件所示。我们为这个错误道歉。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Correction to “Effects of single and combined immunotherapy approach targeting amyloid β protein and α-synuclein in a dementia with Lewy bodies-like model”

Correction to “Effects of single and combined immunotherapy approach targeting amyloid β protein and α-synuclein in a dementia with Lewy bodies-like model”

Correction to “Effects of single and combined immunotherapy approach targeting amyloid β protein and α-synuclein in a dementia with Lewy bodies-like model”

Correction to “Effects of single and combined immunotherapy approach targeting amyloid β protein and α-synuclein in a dementia with Lewy bodies-like model”

Correction to “Effects of single and combined immunotherapy approach targeting amyloid β protein and α-synuclein in a dementia with Lewy bodies-like model”

Mandler M, Rockenstein E, Overk C, et al. Effects of single and combined immunotherapy approach targeting amyloid β protein and α-synuclein in a dementia with Lewy bodies-like model. Alzheimers Dement. 2025;15(9):1133-1148. doi:10.1016/j.jalz.2019.02.002

The authors regret that an error occurred during the assembly of Figure 4G, where the incorrect panels were used to represent the APP/α-syn tg AD02 and the AD02/PD-AFF1 conditions in the S. Nigra, and in Figure 5D, where the incorrect panel was used to represent the APP/α-syn tg AD02/PD-AFF1 in the hippocampus. The corrected figures are provided in the attachment below.  

We apologize for this error.

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来源期刊
Alzheimer's & Dementia
Alzheimer's & Dementia 医学-临床神经学
CiteScore
14.50
自引率
5.00%
发文量
299
审稿时长
3 months
期刊介绍: Alzheimer's & Dementia is a peer-reviewed journal that aims to bridge knowledge gaps in dementia research by covering the entire spectrum, from basic science to clinical trials to social and behavioral investigations. It provides a platform for rapid communication of new findings and ideas, optimal translation of research into practical applications, increasing knowledge across diverse disciplines for early detection, diagnosis, and intervention, and identifying promising new research directions. In July 2008, Alzheimer's & Dementia was accepted for indexing by MEDLINE, recognizing its scientific merit and contribution to Alzheimer's research.
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