{"title":"[更正]活性氧介导的Src -表皮生长因子受体- Akt信号级联激活可阻止硼替佐米诱导的肝癌细胞凋亡。","authors":"Jinlin Hou, Anguo Cui, Peiying Song, Hui Hua, Ting Luo, Yangfu Jiang","doi":"10.3892/mmr.2025.13677","DOIUrl":null,"url":null,"abstract":"<p><p>Following the publication of the above article, the authors have realized that there were several duplicated western blots featured in Figs. 2‑4. The EGFR blot correctly presented for Fig. 3 (the HepG2 cell line) had been inadvertently featured in Fig. 2 (the EGFR blot for the HepG2 cell line). In Fig. 3, the incorrectly assembled Akt blot for the SMMC‑7721 group was the source image for the correctly presented Akt band in Fig. 1 (the SMMC‑7721 cell line), whereas the Akt blot correctly presented for the Hep3B cell line had been inadvertently featured as the actin blot of the HepG2 cell line. The p‑Src blot correctly presented for Fig. 2 (the SMMC‑7721 cell line) had been inadvertently featured in Fig. 4C (the p‑Src blot for SMMC‑7721). Additionally, the actin blots correctly presented for Fig. 2 (the SMMC‑7721 and HepG2 cell lines) were incorrectly assembled into Fig. 4C (the actin blots for SMMC‑7721 and HepG2). The revised versions of Figs. 2‑4, now showing the correct data for the EGFR blot of the HepG2 cell line in Fig. 2, the Akt blot for the SMMC‑7721 cell line and the actin blot for the HepG2 cell line in Fig. 3, the p‑Src blot for the SMMC‑7721 cell line and the actin blots for SMMC 7721 and HepG2 in Fig. 4C, are shown on the next two pages. The authors wish to state that these errors were made during the figure assembly process, and did not affect either the results or the conclusions reported in this paper. All the authors agree with the publication of this corrigendum, and are grateful to the Editor of <i>Molecular Medicine Reports</i> for granting them the opportunity to publish this. The authors regret that these errors were included in the paper, and also apologize to the readership for any inconvenience caused. [Molecular Medicine Reports 11: 712‑718, 2015; DOI: 10.3892/mmr.2014.2736].</p>","PeriodicalId":18818,"journal":{"name":"Molecular medicine reports","volume":"32 6","pages":""},"PeriodicalIF":3.5000,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12446904/pdf/","citationCount":"0","resultStr":"{\"title\":\"[Corrigendum] Reactive oxygen species‑mediated activation of the Src‑epidermal growth factor receptor‑Akt signaling cascade prevents bortezomib‑induced apoptosis in hepatocellular carcinoma cells.\",\"authors\":\"Jinlin Hou, Anguo Cui, Peiying Song, Hui Hua, Ting Luo, Yangfu Jiang\",\"doi\":\"10.3892/mmr.2025.13677\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><p>Following the publication of the above article, the authors have realized that there were several duplicated western blots featured in Figs. 2‑4. The EGFR blot correctly presented for Fig. 3 (the HepG2 cell line) had been inadvertently featured in Fig. 2 (the EGFR blot for the HepG2 cell line). In Fig. 3, the incorrectly assembled Akt blot for the SMMC‑7721 group was the source image for the correctly presented Akt band in Fig. 1 (the SMMC‑7721 cell line), whereas the Akt blot correctly presented for the Hep3B cell line had been inadvertently featured as the actin blot of the HepG2 cell line. The p‑Src blot correctly presented for Fig. 2 (the SMMC‑7721 cell line) had been inadvertently featured in Fig. 4C (the p‑Src blot for SMMC‑7721). Additionally, the actin blots correctly presented for Fig. 2 (the SMMC‑7721 and HepG2 cell lines) were incorrectly assembled into Fig. 4C (the actin blots for SMMC‑7721 and HepG2). The revised versions of Figs. 2‑4, now showing the correct data for the EGFR blot of the HepG2 cell line in Fig. 2, the Akt blot for the SMMC‑7721 cell line and the actin blot for the HepG2 cell line in Fig. 3, the p‑Src blot for the SMMC‑7721 cell line and the actin blots for SMMC 7721 and HepG2 in Fig. 4C, are shown on the next two pages. The authors wish to state that these errors were made during the figure assembly process, and did not affect either the results or the conclusions reported in this paper. All the authors agree with the publication of this corrigendum, and are grateful to the Editor of <i>Molecular Medicine Reports</i> for granting them the opportunity to publish this. The authors regret that these errors were included in the paper, and also apologize to the readership for any inconvenience caused. [Molecular Medicine Reports 11: 712‑718, 2015; DOI: 10.3892/mmr.2014.2736].</p>\",\"PeriodicalId\":18818,\"journal\":{\"name\":\"Molecular medicine reports\",\"volume\":\"32 6\",\"pages\":\"\"},\"PeriodicalIF\":3.5000,\"publicationDate\":\"2025-12-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12446904/pdf/\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Molecular medicine reports\",\"FirstCategoryId\":\"3\",\"ListUrlMain\":\"https://doi.org/10.3892/mmr.2025.13677\",\"RegionNum\":3,\"RegionCategory\":\"医学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"2025/9/12 0:00:00\",\"PubModel\":\"Epub\",\"JCR\":\"Q2\",\"JCRName\":\"MEDICINE, RESEARCH & EXPERIMENTAL\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Molecular medicine reports","FirstCategoryId":"3","ListUrlMain":"https://doi.org/10.3892/mmr.2025.13677","RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"2025/9/12 0:00:00","PubModel":"Epub","JCR":"Q2","JCRName":"MEDICINE, RESEARCH & EXPERIMENTAL","Score":null,"Total":0}
[Corrigendum] Reactive oxygen species‑mediated activation of the Src‑epidermal growth factor receptor‑Akt signaling cascade prevents bortezomib‑induced apoptosis in hepatocellular carcinoma cells.
Following the publication of the above article, the authors have realized that there were several duplicated western blots featured in Figs. 2‑4. The EGFR blot correctly presented for Fig. 3 (the HepG2 cell line) had been inadvertently featured in Fig. 2 (the EGFR blot for the HepG2 cell line). In Fig. 3, the incorrectly assembled Akt blot for the SMMC‑7721 group was the source image for the correctly presented Akt band in Fig. 1 (the SMMC‑7721 cell line), whereas the Akt blot correctly presented for the Hep3B cell line had been inadvertently featured as the actin blot of the HepG2 cell line. The p‑Src blot correctly presented for Fig. 2 (the SMMC‑7721 cell line) had been inadvertently featured in Fig. 4C (the p‑Src blot for SMMC‑7721). Additionally, the actin blots correctly presented for Fig. 2 (the SMMC‑7721 and HepG2 cell lines) were incorrectly assembled into Fig. 4C (the actin blots for SMMC‑7721 and HepG2). The revised versions of Figs. 2‑4, now showing the correct data for the EGFR blot of the HepG2 cell line in Fig. 2, the Akt blot for the SMMC‑7721 cell line and the actin blot for the HepG2 cell line in Fig. 3, the p‑Src blot for the SMMC‑7721 cell line and the actin blots for SMMC 7721 and HepG2 in Fig. 4C, are shown on the next two pages. The authors wish to state that these errors were made during the figure assembly process, and did not affect either the results or the conclusions reported in this paper. All the authors agree with the publication of this corrigendum, and are grateful to the Editor of Molecular Medicine Reports for granting them the opportunity to publish this. The authors regret that these errors were included in the paper, and also apologize to the readership for any inconvenience caused. [Molecular Medicine Reports 11: 712‑718, 2015; DOI: 10.3892/mmr.2014.2736].
期刊介绍:
Molecular Medicine Reports is a monthly, peer-reviewed journal available in print and online, that includes studies devoted to molecular medicine, underscoring aspects including pharmacology, pathology, genetics, neurosciences, infectious diseases, molecular cardiology and molecular surgery. In vitro and in vivo studies of experimental model systems pertaining to the mechanisms of a variety of diseases offer researchers the necessary tools and knowledge with which to aid the diagnosis and treatment of human diseases.