TCRαβ+CD4-CD8- T细胞在牙周炎中的治疗潜力。

IF 5.9 1区 医学 Q1 DENTISTRY, ORAL SURGERY & MEDICINE
L S Ma,Z S Shen,S Y Huang,M Y Li,D Tian,D Zhang,S L Wang
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引用次数: 0

摘要

牙周炎是一种普遍的慢性炎症性疾病,以牙周组织和牙槽骨的进行性退化为特征。尽管对牙周炎发病机制的了解取得了显著进展,但TCRαβ+CD4-CD8- T细胞,也称为双阴性T细胞(DNT)在牙周炎病理生理中的作用尚未得到彻底的研究。在这项研究中,我们观察到与健康个体相比,牙周炎患者牙龈组织中TCRαβ+ DNT细胞的频率显著降低。通过小鼠模型,我们证明了TCRαβ+ DNT细胞的治疗性管理导致牙槽骨吸收减少和炎症生物标志物减少,在较低的细胞剂量下观察到最显著的效果。组织学检查和基因表达分析显示,促炎细胞因子的表达水平明显下降。此外,转录组学分析阐明了与中性粒细胞激活和白细胞介素-17信号通路相关的下调,这在牙周炎的炎症级联反应中至关重要。体外和体内实验都强调了穿孔素在TCRαβ+ DNT细胞中的关键作用,它在调节牙周炎症和防止牙槽骨丢失方面至关重要。总之,我们的研究结果表明,TCRαβ+ DNT细胞治疗可能代表了牙周炎的一种有前景的新治疗策略,为这种普遍口腔健康状况的创新治疗方式的发展提供了有价值的见解。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Therapeutic Potential of TCRαβ+CD4-CD8- T Cells in Periodontitis.
Periodontitis, a pervasive chronic inflammatory disorder, is distinguished by the progressive degradation of periodontal tissues and alveolar bone. Despite remarkable progress in understanding the pathogenesis of periodontitis, the involvement of TCRαβ+CD4-CD8- T cells, also known as double-negative T (DNT) cells, in the pathophysiology of this disease has not been thoroughly investigated. In this study, we observed a significant reduction in the frequency of TCRαβ+ DNT cells within the gingival tissues of patients afflicted with periodontitis when compared with healthy individuals. Employing a murine model, we demonstrated that the therapeutic administration of TCRαβ+ DNT cells resulted in a reduction of alveolar bone resorption and a decrease in inflammatory biomarkers, with the most significant effects observed at lower cell doses. Histological examination and gene expression analysis revealed a notable attenuation in the expression levels of proinflammatory cytokines. Furthermore, transcriptomic profiling elucidated the downregulation of pathways associated with neutrophil activation and interleukin-17 signaling, which are critical in the inflammatory cascade of periodontitis. Both in vitro and in vivo experiments underscored the pivotal role of perforin in TCRαβ+ DNT cells, which is essential for modulating periodontal inflammation and preventing alveolar bone loss. Collectively, our findings suggest that TCRαβ+ DNT cell therapy may represent a promising novel therapeutic strategy for periodontitis, providing valuable insights into the development of innovative treatment modalities for this prevalent oral health condition.
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来源期刊
Journal of Dental Research
Journal of Dental Research 医学-牙科与口腔外科
CiteScore
15.30
自引率
3.90%
发文量
155
审稿时长
3-8 weeks
期刊介绍: The Journal of Dental Research (JDR) is a peer-reviewed scientific journal committed to sharing new knowledge and information on all sciences related to dentistry and the oral cavity, covering health and disease. With monthly publications, JDR ensures timely communication of the latest research to the oral and dental community.
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