CNL蛋白与NRCX形成NLR对,调节植物免疫。

IF 5.8
Xiaohua Dong, Xiaoyan Zhang, Xu Lu, Yufeng Yang, Chuyan Xia, Weiye Pan, Zhiyuan Yin, Yaning Zhao, Gan Ai, Daolong Dou
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引用次数: 0

摘要

核苷酸结合的富含亮氨酸重复序列(NLR)蛋白组装成遗传连锁对,介导植物的效应触发免疫(ETI)。本研究中,我们对本烟中NLRs NRCX和NRCX相邻抗性基因Y的配对进行了表征。CRISPR/Cas9敲除NRCX导致严重的侏儒症和组成性免疫激活,其标志是PR1上调和对辣椒疫霉的抗性增强。共沉默或双敲除邻近NLR的NARY部分挽救了nrx表型,揭示了NARY作为调节生长和免疫的代偿调节因子。结构分析表明,尽管与NLR执行子存在差异,但其包含非规范的Walker B和MHD基序,缺乏自激活能力。分裂荧光素酶和共免疫沉淀实验表明,NRCX和NARY仅通过其CC结构域相互作用,形成非规范调节复合物。值得注意的是,同时沉默NRC2/3和NARY不能完全恢复nrcx突变体的生长,这暗示了免疫调节中的其他因素。我们的研究结果表明,在不引发细胞死亡的情况下,NRCX作为NLR的补偿性伴侣,可以对免疫进行微调,从而揭示了茄科植物平衡生长和防御的新机制。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
A CNL protein forms an NLR pair with NRCX to modulate plant immunity.

Nucleotide-binding leucine-rich repeat (NLR) proteins assemble into genetically linked pairs to mediate effector-triggered immunity (ETI) in plants. Here, we characterize the paired NLRs NRCX and NARY (NRCX adjacent resistance gene Y) in Nicotiana benthamiana. CRISPR/Cas9 knockout of NRCX caused severe dwarfism and constitutively activated immunity, marked by PR1 upregulation and enhanced resistance to Phytophthora capsici. Co-silencing or double knockout of the adjacent NLR NARY partially rescued the nrcx phenotype, revealing NARY as a compensatory regulator that modulates growth and immunity. Structural analysis revealed that NARY harbors non-canonical Walker B and MHD motifs, which lack autoactivation capacity despite their divergence from canonical NLR executors. Split-luciferase and co-immunoprecipitation assays showed that NRCX and NARY interact exclusively through their CC domains, forming a non-canonical regulatory complex. Notably, simultaneous silencing of NRC2/3 and NARY incompletely restored growth in nrcx mutants, implicating additional factors in immune modulation. Our findings establish NARY as a compensatory NLR partner of NRCX that fine-tunes immunity without triggering cell death, revealing a novel mechanism for balancing growth and defense in Solanaceae.

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