勘误表：BMAL1-TTK-H2Bub1环缺乏有助于衰老骨质疏松症中bm - msc介导的骨形成受损。

IF 6.1 2区医学 Q1 MEDICINE, RESEARCH & EXPERIMENTAL

Molecular Therapy. Nucleic Acids Pub Date : 2025-08-13 eCollection Date: 2025-09-09 DOI:10.1016/j.omtn.2025.102638

Li Jinteng, Xu Peitao, Yu Wenhui, Ye Guiwen, Ye Feng, Xu Xiaojun, Su Zepeng, Lin Jiajie, Che Yunshu, Zhang Zhaoqiang, Zeng Yipeng, Li Zhikun, Feng Pei, Cao Qian, Li Dateng, Xie Zhongyu, Wu Yanfeng, Shen Huiyong

引用次数: 0

摘要

[这更正了文章DOI: 10.1016/ j.i omtn.2023.02.014.]。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Erratum: BMAL1-TTK-H2Bub1 loop deficiency contributes to impaired BM-MSC-mediated bone formation in senile osteoporosis.

[This corrects the article DOI: 10.1016/j.omtn.2023.02.014.].

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来源期刊

Molecular Therapy. Nucleic Acids MEDICINE, RESEARCH & EXPERIMENTAL-

CiteScore

15.40

自引率

1.10%

发文量

336

审稿时长

20 weeks

期刊介绍： Molecular Therapy Nucleic Acids is an international, open-access journal that publishes high-quality research in nucleic-acid-based therapeutics to treat and correct genetic and acquired diseases. It is the official journal of the American Society of Gene & Cell Therapy and is built upon the success of Molecular Therapy. The journal focuses on gene- and oligonucleotide-based therapies and publishes peer-reviewed research, reviews, and commentaries. Its impact factor for 2022 is 8.8. The subject areas covered include the development of therapeutics based on nucleic acids and their derivatives, vector development for RNA-based therapeutics delivery, utilization of gene-modifying agents like Zn finger nucleases and triplex-forming oligonucleotides, pre-clinical target validation, safety and efficacy studies, and clinical trials.