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{"title":"撤回声明:生长抑素通过Tollip/髓细胞分化因子88/核因子κ b信号通路减轻急性肠缺血再灌注时肠上皮屏障损伤。","authors":"","doi":"10.1080/21655979.2025.2548109","DOIUrl":null,"url":null,"abstract":"","PeriodicalId":8919,"journal":{"name":"Bioengineered","volume":"16 1","pages":"2548109"},"PeriodicalIF":4.2000,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12396123/pdf/","citationCount":"0","resultStr":"{\"title\":\"Statement of retraction: Somatostatin attenuates intestinal epithelial barrier injury during acute intestinal ischemia-reperfusion through Tollip/Myeloiddifferentiationfactor 88/Nuclear factor kappa-B signaling.\",\"authors\":\"\",\"doi\":\"10.1080/21655979.2025.2548109\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"\",\"PeriodicalId\":8919,\"journal\":{\"name\":\"Bioengineered\",\"volume\":\"16 1\",\"pages\":\"2548109\"},\"PeriodicalIF\":4.2000,\"publicationDate\":\"2025-12-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12396123/pdf/\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Bioengineered\",\"FirstCategoryId\":\"99\",\"ListUrlMain\":\"https://doi.org/10.1080/21655979.2025.2548109\",\"RegionNum\":4,\"RegionCategory\":\"生物学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"2025/8/28 0:00:00\",\"PubModel\":\"Epub\",\"JCR\":\"Q2\",\"JCRName\":\"BIOTECHNOLOGY & APPLIED MICROBIOLOGY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Bioengineered","FirstCategoryId":"99","ListUrlMain":"https://doi.org/10.1080/21655979.2025.2548109","RegionNum":4,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"2025/8/28 0:00:00","PubModel":"Epub","JCR":"Q2","JCRName":"BIOTECHNOLOGY & APPLIED MICROBIOLOGY","Score":null,"Total":0}
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