纤维蛋白水凝胶诱导M2巨噬细胞促进骨再生。

IF 2.9 3区 医学 Q3 CELL & TISSUE ENGINEERING
Ryosuke Aihara, Kazumasa Murata, Tomo Unzai, Chiaki Kitamura, Yasuhiko Tabata
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引用次数: 0

摘要

骨再生仍然是再生医学的一个重大挑战。在此背景下,纤维蛋白水凝胶作为一种有前景的生物材料引起了人们的关注,它可以通过影响巨噬细胞来调节炎症反应并促进组织修复。在这项研究中,我们研究了纤维蛋白水凝胶对巨噬细胞极化的免疫调节作用及其随后对骨再生的影响。人们普遍认为M1巨噬细胞产生肿瘤坏死因子α (TNF-α), M2巨噬细胞产生白细胞介素-10 (IL-10)。当脂多糖(LPS)刺激未分化小鼠骨髓源性巨噬细胞时,观察到促炎细胞因子TNF-α显着增加。然而,在LPS存在的情况下,与纤维蛋白水凝胶共培养可显著抑制TNF-α的产生,同时增强抗炎细胞因子IL-10的分泌。此外,在大鼠颅骨缺损模型中,纤维蛋白水凝胶植入1周后的组织分析显示M2巨噬细胞标记物(CD163、CD204和CD206)上调,表明向抗炎表型转变。值得注意的是,植入后11周,纤维蛋白水凝胶处理的部位表现出增强的骨再生。这些发现强调了纤维蛋白水凝胶作为一种免疫调节生物材料的潜力,它通过促进M2巨噬细胞极化和调节局部炎症微环境来促进骨修复。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Induction of M2 Macrophages by Fibrin Hydrogels Enhances Bone Regeneration.

Bone regeneration remains a significant challenge in regenerative medicine. In this context, fibrin hydrogels have attracted attention as a promising biomaterial that regulates the inflammatory response and promotes tissue repair by influencing macrophages. In this study, we investigated the immunomodulatory effects of fibrin hydrogels on macrophage polarization and their subsequent impact on bone regeneration. It is widely recognized that M1 macrophages produce tumor necrosis factor alpha (TNF-α), while M2 macrophages produce interleukin-10 (IL-10). When undifferentiated mouse bone marrow-derived macrophages were stimulated with lipopolysaccharides (LPS), a marked increase in the proinflammatory cytokine TNF-α was observed. However, coculture with fibrin hydrogels in the presence of LPS significantly suppressed TNF-α production while enhancing the secretion of the anti-inflammatory cytokine IL-10. Furthermore, in a rat calvarial defect model, tissue analysis 1-week postimplantation of fibrin hydrogels revealed an upregulation of M2 macrophage markers (CD163, CD204, and CD206), indicating a shift toward an anti-inflammatory phenotype. Notably, 11 weeks after implantation, the fibrin hydrogel-treated sites exhibited enhanced bone regeneration. These findings highlight the potential of fibrin hydrogels as an immunomodulatory biomaterial that facilitates bone repair by promoting M2 macrophage polarization and modulating the local inflammatory microenvironment.

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来源期刊
Tissue Engineering Part A
Tissue Engineering Part A Chemical Engineering-Bioengineering
CiteScore
9.20
自引率
2.40%
发文量
163
审稿时长
3 months
期刊介绍: Tissue Engineering is the preeminent, biomedical journal advancing the field with cutting-edge research and applications that repair or regenerate portions or whole tissues. This multidisciplinary journal brings together the principles of engineering and life sciences in the creation of artificial tissues and regenerative medicine. Tissue Engineering is divided into three parts, providing a central forum for groundbreaking scientific research and developments of clinical applications from leading experts in the field that will enable the functional replacement of tissues.
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