草酸钙肾结石形成过程中的凋亡、铁下垂、坏死、坏死和焦下垂。

IF 2.2 2区 医学 Q2 UROLOGY & NEPHROLOGY
Saeed R Khan, Abdel A Alli
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引用次数: 0

摘要

肾结石是最常见和衰弱的泌尿系统疾病之一,给医疗保健服务带来了巨大的经济负担。大多数肾结石是由草酸钙和磷酸钙混合而成。其发病机制与肾脏内结晶和晶体滞留有关,这可能是通过在末端收集管中形成晶体塞,阻塞其进入肾盂的开口,或在肾乳头状表面形成磷酸钙斑块实现的。这些栓子被称为兰德尔栓子,而斑块被称为兰德尔斑块。已经进行了一些细胞培养和动物模型研究,以提高我们对草酸钙肾结石发病机制的理解,从而开发出更好的治疗方法。这些研究结果表明,暴露于草酸盐和草酸钙/磷酸盐晶体会导致活性氧的产生和局部损伤和炎症。此外,暴露的细胞有自噬和成骨改变的迹象。损伤和细胞死亡的方式包括凋亡、铁下垂、坏死、坏死下垂和焦下垂。我们对相关文献的回顾表明,坏死和坏死性改变可能涉及兰德尔塞和相关肾结石的形成。兰德尔氏斑块的形成很可能是氧化应激引起的Henle袢和乳头状集管四肢小管上皮成骨改变和MMPs生成的结果。磷酸钙沉积始于基底膜,继续通过间质,矿化胶原蛋白和膜结合囊泡,直到到达乳头表面。尿路上皮的丢失很可能是通过MMPs的激活使斑块暴露于盆腔尿液中。栓子和斑块都是晶体进一步沉积的平台,最终形成结石。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Apoptosis, ferroptosis, necrosis, necroptosis and pyroptosis in the formation of calcium oxalate kidney stones.

Kidney stones are one of the most common and debilitating urological disorders, putting substantial financial burden on healthcare services. Most common kidney stones are comprised of calcium oxalate often mixed with some calcium phosphate. Pathogenesis involves crystallization and retention of crystals within the kidneys, which is achieved either through the formation of crystalline plugs in the terminal collecting ducts blocking their openings into the renal pelvis, or formation of plaques of calcium phosphate on the renal papillary surface. The plugs are termed Randall's plugs and the plaques Randall's plaques. Several cell culture and animal model studies have been carried out to improve our understanding of the pathogenesis of calcium oxalate kidney stones to develop better treatments for the disease. Results of such studies have shown that exposure to oxalate and calcium oxalate/phosphate crystal leads to the production of reactive oxygen species and localized injury and inflammation. In addition, there are signs of autophagy and osteogenic changes in exposed cells. Modes of injury and cell death include apoptosis, ferroptosis, necrosis, necroptosis, and pyroptosis. Our review of relevant literature indicates that necrotic and necroptotic changes may be involved in the formation of Randall's plugs and associated kidney stones. Randall's plaque formation is most likely an outcome of the oxidant stress induced osteogenic changes in the tubular epithelium of the limbs of the loops of Henle and papillary collecting ducts and production of MMPs. Calcium phosphate deposition starts in the basement membrane, continues through the interstitium, mineralizing the collagen and membrane bound vesicles, until it reaches the papillary surface. The loss of urothelium most likely through the activation of MMPs exposes the plaque to the pelvic urine. Both plugs and plaque act as the platform for further deposition of crystals eventually developing into the stones.

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来源期刊
Urolithiasis
Urolithiasis UROLOGY & NEPHROLOGY-
CiteScore
4.50
自引率
6.50%
发文量
74
期刊介绍: Official Journal of the International Urolithiasis Society The journal aims to publish original articles in the fields of clinical and experimental investigation only within the sphere of urolithiasis and its related areas of research. The journal covers all aspects of urolithiasis research including the diagnosis, epidemiology, pathogenesis, genetics, clinical biochemistry, open and non-invasive surgical intervention, nephrological investigation, chemistry and prophylaxis of the disorder. The Editor welcomes contributions on topics of interest to urologists, nephrologists, radiologists, clinical biochemists, epidemiologists, nutritionists, basic scientists and nurses working in that field. Contributions may be submitted as full-length articles or as rapid communications in the form of Letters to the Editor. Articles should be original and should contain important new findings from carefully conducted studies designed to produce statistically significant data. Please note that we no longer publish articles classified as Case Reports. Editorials and review articles may be published by invitation from the Editorial Board. All submissions are peer-reviewed. Through an electronic system for the submission and review of manuscripts, the Editor and Associate Editors aim to make publication accessible as quickly as possible to a large number of readers throughout the world.
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