靶向转移性富马酸水合酶缺乏肾细胞癌的代谢和表观遗传重编程。

IF 3.2 3区 医学 Q2 ONCOLOGY
Nadja Kührer, Irene Huebner-Resch, Roman Mayr, Abbas Agaimy, Andreas Kronbichler, Arndt Hartmann, Manuela Schmidinger, Renate Pichler
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引用次数: 0

摘要

富马酸水合酶缺陷型肾细胞癌(FHdRCC)已根据世卫组织分类2022被归为分子定义的rcc新类别。虽然罕见,但FHdRCC是一种侵袭性恶性肿瘤,具有高转移潜力和预后差,即使在早期也是如此。由于发病率低,至今尚无标准的治疗方案。一些2期临床试验正在评估针对晚期/转移性疾病患者的联合靶向治疗。这些包括免疫检查点抑制剂(nivolumab, tislelizumab, sintilimab, avelumab),多酪氨酸激酶抑制剂(cabozantinib, erlotinib, lenvatinib, axitinib, vandetanib)和PARP抑制剂(talazoparib)。最有希望的组合是尼武单抗/卡博赞替尼(N = 5,客观缓解率(ORR): 100%)、lenvatinib/tislelizumab (N = 14, ORR: 93.3%)、贝伐单抗/erlotinib (N = 43, ORR: 72%)和sintilimab/axitinib (N = 19, ORR: 63.1%)。在这篇综述中,我们将提供正在进行的临床试验的详细概述,强调代谢和表观遗传重编程的作用,以及致癌前信号,它们共同构成了新兴的靶向治疗策略的支柱。针对这些特定的信号通路将使转移性FHdRCC的治疗前景转向个性化治疗。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Targeting metabolic and epigenetic reprogramming in metastatic fumarate hydratase-deficient renal cell carcinoma.

Targeting metabolic and epigenetic reprogramming in metastatic fumarate hydratase-deficient renal cell carcinoma.

Targeting metabolic and epigenetic reprogramming in metastatic fumarate hydratase-deficient renal cell carcinoma.

Targeting metabolic and epigenetic reprogramming in metastatic fumarate hydratase-deficient renal cell carcinoma.

Fumarate hydratase-deficient renal cell carcinoma (FHdRCC) has been classified under the new category of molecularly defined RCCs according to the WHO classification 2022. Although rare, FHdRCC is an aggressive malignancy with a high metastatic potential and poor prognosis, even at early stages. Due to its low incidence, no standard therapeutic regimen has been established to date. Several phase 2 clinical trials are evaluating combinations of targeted therapies in patients with advanced/metastatic disease. These include immune checkpoint inhibitors (nivolumab, tislelizumab, sintilimab, avelumab), multi-tyrosine kinase inhibitors (cabozantinib, erlotinib, lenvatinib, axitinib, vandetanib), and PARP inhibitors (talazoparib). The most promising combinations are nivolumab/cabozantinib (N = 5, objective response rate (ORR): 100%), lenvatinib/tislelizumab (N = 14, ORR: 93.3%), bevacizumab/erlotinib (N = 43, ORR: 72%), and sintilimab/axitinib (N = 19, ORR: 63.1%). In this review, we will provide a detailed overview of ongoing clinical trials, highlighting the roles of metabolic and epigenetic reprogramming, as well as pro- oncogenic signaling, which together form the backbone for emerging novel targeted treatment strategies. Targeting these specific signaling pathways will shift the therapeutic landscape toward personalized medicine in metastatic FHdRCC.

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来源期刊
CiteScore
7.80
自引率
5.00%
发文量
55
审稿时长
12 months
期刊介绍: The Journal''s scope encompasses all aspects of metastasis research, whether laboratory-based, experimental or clinical and therapeutic. It covers such areas as molecular biology, pharmacology, tumor biology, and clinical cancer treatment (with all its subdivisions of surgery, chemotherapy and radio-therapy as well as pathology and epidemiology) insofar as these disciplines are concerned with the Journal''s core subject of metastasis formation, prevention and treatment.
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