以连接分子Kirrel3为标志的抑制性神经元调节记忆精度。

IF 4 2区 医学 Q1 NEUROSCIENCES
Arnulfo Tuñon-Ortiz, Dimitri Tränkner, Chandler M Peterson, Omar Shennib, Fangfei Ye, Jiani Shi, Sarah N Brockway, Olivia Raines, Abbey Mahnke, Matthew Grega, Keun-Young Kim, Mark H Ellisman, James G Heys, Moriel Zelikowsky, Megan E Williams
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引用次数: 0

摘要

亲同性粘附分子Kirrel3驱动齿状颗粒(DG)神经元和GABA神经元之间的突触形成,Kirrel3基因变异与人类神经发育障碍有关。然而,表达kirrel3的神经元的电路功能和行为相关性尚不清楚。利用交叉遗传学,我们鉴定了一群表达kirrel3的GABA神经元,这些神经元调节雄性和雌性小鼠的记忆区分。利用化学遗传学、体内电生理学和行为分析,我们发现激活表达kirrel3的GABA神经元,而不是小白蛋白神经元,可以有效抑制CA3神经元的活动,并在回忆过程中损害情境记忆辨别,揭示了这些神经元在精确记忆检索中的关键作用。光镜和电镜显示,表达kirrel3的GABA神经元受到DG神经元的直接激发,并投射到CA3树突上。总之,这种多尺度方法展示了粘附分子的细胞类型特异性表达如何标记控制指导记忆和行为的关键特征的神经元子集。在这里,我们发现了一组从未被研究过的由基因Kirrel3表达定义的抑制性神经元,Kirrel3是一种突触细胞粘附分子和神经发育障碍的风险基因。我们发现,Kirrel3抑制神经元活动对CA3神经元活动施加了异常强烈的控制,并损害了小鼠在记忆检索过程中区分不同上下文的能力。因此,Kirrel3抑制神经元调节小鼠的记忆精度,我们的研究采用了一个强大的框架,将分子身份和突触特异性与成人大脑的行为功能联系起来。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Inhibitory Neurons Marked by the Connectivity Molecule Kirrel3 Regulate Memory Precision.

The homophilic adhesion molecule Kirrel3 drives synapse formation between dentate granule (DG) neurons and GABA neurons, and Kirrel3 gene variants are associated with neurodevelopmental disorders in humans. However, the circuit function and behavioral relevance of Kirrel3-expressing neurons are unknown. Using intersectional genetics, we identified a population of Kirrel3-expressing GABA neurons that regulate memory discrimination in male and female mice. Using chemogenetics with in vivo electrophysiology and behavioral assays, we discovered that activating Kirrel3-expressing GABA neurons, but not parvalbumin neurons, potently inhibits CA3 neuron activity and impairs contextual memory discrimination during recall, revealing a critical role for these neurons in the retrieval of precise memories. Light and electron microscopy of Kirrel3-expressing GABA neurons suggests that they receive direct excitation from DG neurons and project onto CA3 dendrites. Together, this multiscale approach demonstrates how cell type-specific expression of adhesion molecules mark subsets of neurons that control key features guiding memory and behavior.

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来源期刊
Journal of Neuroscience
Journal of Neuroscience 医学-神经科学
CiteScore
9.30
自引率
3.80%
发文量
1164
审稿时长
12 months
期刊介绍: JNeurosci (ISSN 0270-6474) is an official journal of the Society for Neuroscience. It is published weekly by the Society, fifty weeks a year, one volume a year. JNeurosci publishes papers on a broad range of topics of general interest to those working on the nervous system. Authors now have an Open Choice option for their published articles
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