解开精神障碍之谜:遗传学优先的方法和基于罕见疾病易感基因组变异的小鼠模型的作用。

IF 0.9 4区 医学 Q4 MEDICINE, RESEARCH & EXPERIMENTAL
Reon Kondo, Daisuke Mori, Hiroaki Wake, Norio Ozaki
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引用次数: 0

摘要

精神障碍是全球致残的主要原因,涉及思维、情绪调节或行为方面的重大障碍。这些疾病的发病机制由于其模糊的性质和缺乏生物标志物而变得复杂。人们提出了一种遗传学优先的方法来解决这种复杂性。该方法将临床表型与疾病易感基因组变异(如拷贝数变异和单核苷酸变异)联系起来。这些罕见的变异显著影响疾病的发展,因此对于评估特定变异对疾病的影响和确定潜在的生物学机制至关重要。特别是,反映这些变异的小鼠模型有助于确定遗传变异与疾病相关表型之间的因果关系。最近的研究强调了人类和小鼠感觉信息处理的重要性。在揭示这些表型的神经回路机制方面有价值的先进技术包括光遗传学和体内双光子成像。此外,小鼠模型可以指导患者和诱导多能干细胞的研究结果的整合,支持多维方法来理解精神障碍的病理生理。在这篇综述中,我们简要地讨论了小鼠模型在遗传学优先的方法中阐明精神障碍的病理生理的效用。我们还介绍了基于罕见疾病易感变异的小鼠模型的例子。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Unraveling the enigma of mental disorders: a genetics-first approach and the role of mouse models based on rare disease-susceptible genome variants.

Unraveling the enigma of mental disorders: a genetics-first approach and the role of mouse models based on rare disease-susceptible genome variants.

Mental disorders are a major global cause of disability that involve significant disturbances in thinking, emotional regulation, or behavior. The pathogenesis of these illnesses is complicated by their obscure nature and lack of biological markers. A genetics-first approach has been proposed to address this complexity. This approach associates clinical phenotypes with disease-susceptible genomic variants, such as copy number variations and single nucleotide variants. These rare variants significantly affect disease development and are thus crucial for assessing the effects of specific variants on disease and in determining the underlying biological mechanisms. In particular, mouse models that reflect these variants are instrumental in defining the causal relationships between genetic variants and disease-relevant phenotypes. Recent studies have highlighted the importance of sensory information processing in humans and mice. Advanced technologies that are valuable in unraveling the neural circuit mechanisms of these phenotypes include optogenetics and in vivo 2-photon imaging. Furthermore, mouse models can guide the integration of findings from patients and induced pluripotent stem cells, supporting a multidimensional approach to understanding the pathophysiology of mental disorders. In this review, we briefly discuss the utility of mouse models in a genetics-first approach to elucidate the pathophysiology of mental disorders. We also present examples of our mouse models based on rare disease-susceptible variants.

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来源期刊
Nagoya Journal of Medical Science
Nagoya Journal of Medical Science MEDICINE, RESEARCH & EXPERIMENTAL-
CiteScore
1.30
自引率
0.00%
发文量
65
审稿时长
>12 weeks
期刊介绍: The Journal publishes original papers in the areas of medical science and its related fields. Reviews, symposium reports, short communications, notes, case reports, hypothesis papers, medical image at a glance, video and announcements are also accepted. Manuscripts should be in English. It is recommended that an English check of the manuscript by a competent and knowledgeable native speaker be completed before submission.
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