{"title":"更正“蛋白酪氨酸磷酸酶1B通过促进内质网应激,加重NF-κB和胶质细胞激活介导的神经炎症,从而导致神经性疼痛”","authors":"","doi":"10.1111/cns.70557","DOIUrl":null,"url":null,"abstract":"<p>Jiao, B., Zhang, W., Zhang, C., et al. “Protein Tyrosine Phosphatase 1B Contributes to Neuropathic Pain by Aggravating NF-κB and Glial Cells Activation-Mediated Neuroinflammation via Promoting Endoplasmic Reticulum Stress,” <i>CNS Neuroscience & Therapeutics</i> 30, no. 2 (2024): e14609. https://doi.org/10.1111/cns.14609.</p><p>In the original version of this article, there was an error in Figure 2A. Specifically, the representative electron microscopy image for the Sham group was incorrect. The correct image is provided below. The correction does not change the results or conclusions of this paper.</p>","PeriodicalId":154,"journal":{"name":"CNS Neuroscience & Therapeutics","volume":"31 8","pages":""},"PeriodicalIF":5.0000,"publicationDate":"2025-08-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/cns.70557","citationCount":"0","resultStr":"{\"title\":\"Correction to “Protein Tyrosine Phosphatase 1B Contributes to Neuropathic Pain by Aggravating NF-κB and Glial Cells Activation-Mediated Neuroinflammation via Promoting Endoplasmic Reticulum Stress”\",\"authors\":\"\",\"doi\":\"10.1111/cns.70557\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>Jiao, B., Zhang, W., Zhang, C., et al. “Protein Tyrosine Phosphatase 1B Contributes to Neuropathic Pain by Aggravating NF-κB and Glial Cells Activation-Mediated Neuroinflammation via Promoting Endoplasmic Reticulum Stress,” <i>CNS Neuroscience & Therapeutics</i> 30, no. 2 (2024): e14609. https://doi.org/10.1111/cns.14609.</p><p>In the original version of this article, there was an error in Figure 2A. Specifically, the representative electron microscopy image for the Sham group was incorrect. The correct image is provided below. The correction does not change the results or conclusions of this paper.</p>\",\"PeriodicalId\":154,\"journal\":{\"name\":\"CNS Neuroscience & Therapeutics\",\"volume\":\"31 8\",\"pages\":\"\"},\"PeriodicalIF\":5.0000,\"publicationDate\":\"2025-08-04\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1111/cns.70557\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"CNS Neuroscience & Therapeutics\",\"FirstCategoryId\":\"3\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1111/cns.70557\",\"RegionNum\":1,\"RegionCategory\":\"医学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q1\",\"JCRName\":\"NEUROSCIENCES\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"CNS Neuroscience & Therapeutics","FirstCategoryId":"3","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1111/cns.70557","RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"NEUROSCIENCES","Score":null,"Total":0}
Correction to “Protein Tyrosine Phosphatase 1B Contributes to Neuropathic Pain by Aggravating NF-κB and Glial Cells Activation-Mediated Neuroinflammation via Promoting Endoplasmic Reticulum Stress”
Jiao, B., Zhang, W., Zhang, C., et al. “Protein Tyrosine Phosphatase 1B Contributes to Neuropathic Pain by Aggravating NF-κB and Glial Cells Activation-Mediated Neuroinflammation via Promoting Endoplasmic Reticulum Stress,” CNS Neuroscience & Therapeutics 30, no. 2 (2024): e14609. https://doi.org/10.1111/cns.14609.
In the original version of this article, there was an error in Figure 2A. Specifically, the representative electron microscopy image for the Sham group was incorrect. The correct image is provided below. The correction does not change the results or conclusions of this paper.
期刊介绍:
CNS Neuroscience & Therapeutics provides a medium for rapid publication of original clinical, experimental, and translational research papers, timely reviews and reports of novel findings of therapeutic relevance to the central nervous system, as well as papers related to clinical pharmacology, drug development and novel methodologies for drug evaluation. The journal focuses on neurological and psychiatric diseases such as stroke, Parkinson’s disease, Alzheimer’s disease, depression, schizophrenia, epilepsy, and drug abuse.