{"title":"更正“Pla2g5通过巨噬细胞增殖和LA/Ffar1肺细胞募集参与病毒样诱导的肺部炎症”。","authors":"","doi":"10.1111/imm.70017","DOIUrl":null,"url":null,"abstract":"<p>M. Koganesawa, D. F. Dwyer, K. Alhallak, J. Nagai, K. Zaleski, S. Samuchiwal, et al., “Pla2g5 Contributes to Viral-Like-Induced Lung Inflammation Through Macrophage Proliferation and LA/Ffar1 Lung Cell Recruitment,” <i>Immunology</i> 172, no. 1 (2024): 144–162, https://doi.org/10.1111/imm.13766.</p><p>In the article, there are labeling errors in Figures 1, 6 and 7.</p><p>We apologise for these errors.</p>","PeriodicalId":13508,"journal":{"name":"Immunology","volume":"176 3","pages":"403-406"},"PeriodicalIF":5.0000,"publicationDate":"2025-07-24","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/imm.70017","citationCount":"0","resultStr":"{\"title\":\"Correction to “Pla2g5 Contributes to Viral-Like-Induced Lung Inflammation Through Macrophage Proliferation and LA/Ffar1 Lung Cell Recruitment”\",\"authors\":\"\",\"doi\":\"10.1111/imm.70017\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>M. Koganesawa, D. F. Dwyer, K. Alhallak, J. Nagai, K. Zaleski, S. Samuchiwal, et al., “Pla2g5 Contributes to Viral-Like-Induced Lung Inflammation Through Macrophage Proliferation and LA/Ffar1 Lung Cell Recruitment,” <i>Immunology</i> 172, no. 1 (2024): 144–162, https://doi.org/10.1111/imm.13766.</p><p>In the article, there are labeling errors in Figures 1, 6 and 7.</p><p>We apologise for these errors.</p>\",\"PeriodicalId\":13508,\"journal\":{\"name\":\"Immunology\",\"volume\":\"176 3\",\"pages\":\"403-406\"},\"PeriodicalIF\":5.0000,\"publicationDate\":\"2025-07-24\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1111/imm.70017\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Immunology\",\"FirstCategoryId\":\"3\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1111/imm.70017\",\"RegionNum\":3,\"RegionCategory\":\"医学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q2\",\"JCRName\":\"IMMUNOLOGY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Immunology","FirstCategoryId":"3","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1111/imm.70017","RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q2","JCRName":"IMMUNOLOGY","Score":null,"Total":0}
Correction to “Pla2g5 Contributes to Viral-Like-Induced Lung Inflammation Through Macrophage Proliferation and LA/Ffar1 Lung Cell Recruitment”
M. Koganesawa, D. F. Dwyer, K. Alhallak, J. Nagai, K. Zaleski, S. Samuchiwal, et al., “Pla2g5 Contributes to Viral-Like-Induced Lung Inflammation Through Macrophage Proliferation and LA/Ffar1 Lung Cell Recruitment,” Immunology 172, no. 1 (2024): 144–162, https://doi.org/10.1111/imm.13766.
In the article, there are labeling errors in Figures 1, 6 and 7.
期刊介绍:
Immunology is one of the longest-established immunology journals and is recognised as one of the leading journals in its field. We have global representation in authors, editors and reviewers.
Immunology publishes papers describing original findings in all areas of cellular and molecular immunology. High-quality original articles describing mechanistic insights into fundamental aspects of the immune system are welcome. Topics of interest to the journal include: immune cell development, cancer immunology, systems immunology/omics and informatics, inflammation, immunometabolism, immunology of infection, microbiota and immunity, mucosal immunology, and neuroimmunology.
The journal also publishes commissioned review articles on subjects of topical interest to immunologists, and commissions in-depth review series: themed sets of review articles which take a 360° view of select topics at the heart of immunological research.