{"title":"更正“gtp结合抑制剂增加lrrk2连接的泛素化和路易体样包涵体”","authors":"","doi":"10.1002/jcp.70057","DOIUrl":null,"url":null,"abstract":"<p>Thomas, J. M., Wang, X., Guo, G., et al. 2020. “GTP-Binding Inhibitors Increase LRRK2-Linked Ubiquitination and Lewy Body-Like Inclusions.” <i>Journal of Cellular Physiology</i> 235: 7309–7320.</p><p>The authors, by reviewing the records of the original western blots from this study, recently found that the anti-Flag (tagged LRRK2) blot in Figure 3A (the middle blot of right panel) had been duplicated in Figure 2A (the middle blot of right panel). It has been discovered that the wrong image was inadvertently selected for Figure 2A due to the similarity in the cropped blots. The authors have located the correct blot image for Figure 2A and provided the corrected Figure 2. The conclusions of this paper are not affected by this error.</p><p>The authors apologize for this error and any confusion.</p>","PeriodicalId":15220,"journal":{"name":"Journal of Cellular Physiology","volume":"240 7","pages":""},"PeriodicalIF":4.0000,"publicationDate":"2025-07-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcp.70057","citationCount":"0","resultStr":"{\"title\":\"Correction to “GTP-Binding Inhibitors Increase LRRK2-Linked Ubiquitination and Lewy Body-Like Inclusions”\",\"authors\":\"\",\"doi\":\"10.1002/jcp.70057\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>Thomas, J. M., Wang, X., Guo, G., et al. 2020. “GTP-Binding Inhibitors Increase LRRK2-Linked Ubiquitination and Lewy Body-Like Inclusions.” <i>Journal of Cellular Physiology</i> 235: 7309–7320.</p><p>The authors, by reviewing the records of the original western blots from this study, recently found that the anti-Flag (tagged LRRK2) blot in Figure 3A (the middle blot of right panel) had been duplicated in Figure 2A (the middle blot of right panel). It has been discovered that the wrong image was inadvertently selected for Figure 2A due to the similarity in the cropped blots. The authors have located the correct blot image for Figure 2A and provided the corrected Figure 2. The conclusions of this paper are not affected by this error.</p><p>The authors apologize for this error and any confusion.</p>\",\"PeriodicalId\":15220,\"journal\":{\"name\":\"Journal of Cellular Physiology\",\"volume\":\"240 7\",\"pages\":\"\"},\"PeriodicalIF\":4.0000,\"publicationDate\":\"2025-07-17\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcp.70057\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of Cellular Physiology\",\"FirstCategoryId\":\"99\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1002/jcp.70057\",\"RegionNum\":2,\"RegionCategory\":\"生物学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q2\",\"JCRName\":\"CELL BIOLOGY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Cellular Physiology","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/jcp.70057","RegionNum":2,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q2","JCRName":"CELL BIOLOGY","Score":null,"Total":0}
Correction to “GTP-Binding Inhibitors Increase LRRK2-Linked Ubiquitination and Lewy Body-Like Inclusions”
Thomas, J. M., Wang, X., Guo, G., et al. 2020. “GTP-Binding Inhibitors Increase LRRK2-Linked Ubiquitination and Lewy Body-Like Inclusions.” Journal of Cellular Physiology 235: 7309–7320.
The authors, by reviewing the records of the original western blots from this study, recently found that the anti-Flag (tagged LRRK2) blot in Figure 3A (the middle blot of right panel) had been duplicated in Figure 2A (the middle blot of right panel). It has been discovered that the wrong image was inadvertently selected for Figure 2A due to the similarity in the cropped blots. The authors have located the correct blot image for Figure 2A and provided the corrected Figure 2. The conclusions of this paper are not affected by this error.
The authors apologize for this error and any confusion.
期刊介绍：
The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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