转录因子TFII-I导致前列腺癌细胞中GRP78合成的转录上调

IF 2.8 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
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引用次数: 0

摘要

引用本文:U. K. Misra, Wang F., S. V. Pizzo,“转录因子TFII-I - i导致前列腺癌细胞GRP78合成的转录上调”,《细胞生物化学杂志》,第106期。3 (2009): 381-389, https://doi.org/10.1002/jcb.22016.The上述文章于2008年12月18日在线发表在Wiley在线图书馆(wileyonlinelibrary.com)上,经期刊主编Christian Behl;和Wiley期刊有限责任公司。由于第三方提出的担忧,已同意撤回。具体来说,在图1A和图3之间以及图3内检测到图像元素的重复。在所有情况下,这些图像元素被用来表示不同的实验条件。一项机构审查证实了这些担忧的有效性。因此,由于编辑对整个数据的准确性和完整性失去信任,并认为文章的结论无效,文章被撤回。作者已被告知撤稿的决定。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
RETRACTION: Transcription Factor TFII-I Causes Transcriptional Upregulation of GRP78 Synthesis in Prostate Cancer Cells

RETRACTION: U. K. Misra, F. Wang, and S. V. Pizzo, “Transcription Factor TFII-I Causes Transcriptional Upregulation of GRP78 Synthesis in Prostate Cancer Cells,” Journal of Cellular Biochemistry 106, no. 3 (2009): 381-389, https://doi.org/10.1002/jcb.22016.

The above article, published online on 18 December 2008 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Christian Behl; and Wiley Periodicals LLC. The retraction has been agreed due to concerns raised by third parties. Specifically, duplication of image elements has been detected between Figures 1A and 3, and within Figure 3. In all instances, these image elements have been used to represent different experimental conditions. An institutional review confirmed the validity of the concerns. Accordingly, the article is retracted as the editors have lost trust in the accuracy and integrity of the whole body of data and consider the conclusions of the article invalid. The authors have been informed of the decision of retraction.

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来源期刊
Journal of cellular biochemistry
Journal of cellular biochemistry 生物-生化与分子生物学
CiteScore
9.90
自引率
0.00%
发文量
164
审稿时长
1 months
期刊介绍: The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.
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