AZD1080是GSK - 3β的特异性抑制剂,可抑制骨肉瘤癌症干细胞样细胞的干性和恶性肿瘤。

IF 3.5 3区 医学 Q2 MEDICINE, RESEARCH & EXPERIMENTAL
Molecular medicine reports Pub Date : 2025-09-01 Epub Date: 2025-07-11 DOI:10.3892/mmr.2025.13613
Peiyu Guo, Zhenkai Lou, Hongda Gong, Xiaodong Hou, Chunqiang Zhang, Bing Wang, Kaili Du
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引用次数: 0

摘要

骨肉瘤是最常见的原发性骨癌,主要影响儿童和年轻人。肿瘤干细胞(CSCs)是一个具有干性的亚群,对预后有重要影响,并促进复发和转移。由于糖原合成酶激酶- 3β (GSK - 3β)在维持干性方面的关键作用,它被认为是药物开发的重要靶点。本研究的目的是评估GSK‑3β抑制剂AZD1080对骨肉瘤CSCs的抑制作用。AZD1080处理明显抑制U2OS和143B细胞的球体形成,并抑制U2OS和143B来源的CSCs的解离球体;在这两个过程中,茎干标志物OCT4和SOX2均显著降低,但不影响细胞增殖和凋亡。AZD1080处理抑制GSK‑3β及其下游调控基因的磷酸化,包括HEY1、HES1、CyclinD1和β‑catenin。我们还观察到,GSK‑3β活性是AZD1080处理对球型形成和茎干性的抑制作用的关键。此外,GSK‑3β敲低抑制了球的形成和侵袭能力,表明AZD1080以GSK‑3β依赖的方式发挥抑制作用。综上所述,AZD1080是一种特异性的CSC干细胞抑制剂,无细胞毒性,表明它是一种有前景的靶向骨肉瘤GSK‑3β信号通路的治疗药物。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
AZD1080, a specific inhibitor of GSK‑3β, inhibits stemness and malignancies in osteosarcoma cancer stem‑like cells.

Osteosarcoma is the most common primary bone cancer, primarily affecting children and young adults. Cancer stem cells (CSCs), a subpopulation presenting stemness, critically influence prognosis and promote recurrence and metastasis. Due to the crucial role of glycogen synthase kinase‑3 beta (GSK‑3β) in maintaining stemness, it is considered as an important target for drug development. The aim of the present study was to evaluate the inhibitory effect of AZD1080, a GSK‑3β inhibitor, on osteosarcoma CSCs. AZD1080 treatment clearly inhibited sphere formation in U2OS and 143B cells and dissociated spheres in CSCs derived from U2OS and 143B; in both processes, stemness markers OCT4 and SOX2 were markedly decreased, without affecting cell proliferation or apoptosis. AZD1080 treatment inhibited phosphorylation of GSK‑3β and its downstream regulated genes, including HEY1, HES1, CyclinD1 and β‑catenin. It was also observed that GSK‑3β activity was critical for the inhibitory effects of AZD1080 treatment on sphere formation and stemness. Moreover, GSK‑3β knockdown inhibited sphere formation and invasion capacity, indicating that AZD1080 exerts inhibitory roles in a GSK‑3β‑dependent manner. Taken together, the results showed AZD1080 as a specific inhibitor of CSC stemness, without cytotoxicity, and indicated it was a promising therapeutic agent that targeted GSK‑3β signaling in osteosarcoma.

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来源期刊
Molecular medicine reports
Molecular medicine reports 医学-病理学
CiteScore
7.60
自引率
0.00%
发文量
321
审稿时长
1.5 months
期刊介绍: Molecular Medicine Reports is a monthly, peer-reviewed journal available in print and online, that includes studies devoted to molecular medicine, underscoring aspects including pharmacology, pathology, genetics, neurosciences, infectious diseases, molecular cardiology and molecular surgery. In vitro and in vivo studies of experimental model systems pertaining to the mechanisms of a variety of diseases offer researchers the necessary tools and knowledge with which to aid the diagnosis and treatment of human diseases.
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