从种群生物库数据中提取和校准变异致病性证据。

IF 8.1 1区 生物学 Q1 GENETICS & HEREDITY
Vineel Bhat, Tian Yu, Lara Brown, Vikas Pejaver, Matthew Lebo, Steven Harrison, Christopher A Cassa
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引用次数: 0

摘要

基因组医学需要一个关于变异表型影响的强有力的证据基础,即使在与单基因疾病相关的广泛研究基因中,这一证据基础仍然不完整。在这里,我们评估了使用人群队列数据来识别可用于变异评估的证据的广泛潜力。研究人员利用469,803名UK Biobank参与者的数据,计算了与18种临床可操作单基因表型相关的41个基因的疾病富集变异水平优势比。我们发现clinvar标记的11种表型的致病变异和良性变异的比值比值存在显著差异,涵盖常见和罕见疾病。为了便于临床翻译,我们校准了可变水平优势比提供的证据强度,以与美国医学遗传学与基因组学学院和分子病理学协会(ACMG/AMP)解释指南(PS4标准)保持一致,并发现优势比可能达到“中等”,“强”或“非常强”的证据,因表型和基因而异。总的来说,我们发现2.6% (N = 12,350)的参与者携带一种罕见的不确定意义变异(VUS),至少有中等程度的致病性证据,这是一种潜在的未被识别的疾病风险的指示。最后,通过结合计算和功能数据以及基于人群的优势比,我们确定了符合临床再分类标准的变异。值得注意的是,使用这种方法,我们发现参与者中12.4%的LDLR罕见VUSs符合诊断标准,被归类为可能致病的,这表明它有可能扩大VUSs的重新分类。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Extracting and calibrating evidence of variant pathogenicity from population biobank data.

Genomic medicine requires a robust evidence base of variant phenotypic impacts, which remains incomplete even in extensively studied genes with monogenic disease associations. Here, we evaluated the broad potential of using population cohort data to identify evidence that can be used in variant assessment. Across 41 genes related to 18 clinically actionable monogenic phenotypes, we calculated variant-level odds ratios of disease enrichment using data from 469,803 UK Biobank participants. We found significant differences in odds ratio values between ClinVar-labeled pathogenic and benign variants in 11 phenotypes, spanning both common and rare disorders. To facilitate clinical translation, we calibrated the strength of evidence provided by variant-level odds ratios to align with American College of Medical Genetics and Genomics and the Association for Molecular Pathology (ACMG/AMP) interpretation guidelines (PS4 criterion) and found that odds ratios may reach "moderate," "strong," or "very strong" evidence, varying by phenotype and gene. Overall, we found that 2.6% (N = 12,350) of participants harbor a rare variant of uncertain significance (VUS) with at least moderate evidence of pathogenicity-an indication of potentially unrecognized disease risk. Finally, by incorporating computational and functional data alongside population-based odds ratios, we identified variants that met the criteria for clinical reclassification. Notably, using this approach, we identified that 12.4% of rare VUSs in LDLR seen in participants meet diagnostic criteria to be classified as likely pathogenic, demonstrating its potential to scale the reclassification of VUSs.

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来源期刊
CiteScore
14.70
自引率
4.10%
发文量
185
审稿时长
1 months
期刊介绍: The American Journal of Human Genetics (AJHG) is a monthly journal published by Cell Press, chosen by The American Society of Human Genetics (ASHG) as its premier publication starting from January 2008. AJHG represents Cell Press's first society-owned journal, and both ASHG and Cell Press anticipate significant synergies between AJHG content and that of other Cell Press titles.
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