修正食品强化在预防神经管缺陷方面的无效，原因是监管促进叶酸和维生素B12水平不足

IF 1.6 4区医学 Q4 DEVELOPMENTAL BIOLOGY

Birth Defects Research Pub Date : 2025-07-11 DOI:10.1002/bdr2.2510

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引用次数: 0

摘要

Kancherla， V.， P. Bhalla， S. K. Dutta， R. Mehta， R. M. Vora和S. Karmarkar, 2025。“在印度，由于监管规定导致叶酸和维生素B12水平不足，食品强化在预防神经管缺陷方面无效。”出生缺陷研究117，no。7: e2498。https://doi.org/10.1002/bdr2.2498.In在最初发表的文章中，第1节给出了一个错误的度量单位。修正后的单位如下所示。这在文章的在线版本中已被更正。对于非孕妇、儿童和男性，2020年推荐的维生素B12日摄食量在1.2 - 2.5毫克之间（FSSAI 2021）。对非孕妇、儿童和男性，2020年推荐的维生素B12日摄食量在1.2至2.5微克之间（FSSAI 2021）。我们为这个错误道歉。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Correction to Food Fortification Ineffective in Preventing Neural Tube Defects in India due to Regulation Promoting Inadequate Levels of Folic Acid and Vitamin B12

Kancherla, V., P. Bhalla, S. K. Dutta, R. Mehta, R. M. Vora, and S. Karmarkar. 2025. “Food Fortification Ineffective in Preventing Neural Tube Defects in India due to Regulation Promoting Inadequate Levels of Folic Acid and Vitamin B12.” Birth Defects Research 117, no. 7: e2498. https://doi.org/10.1002/bdr2.2498.

In the originally published article, an incorrect unit of measurement was given in Section 1. The corrected unit is given below. This has been corrected in the online version of the article.

Incorrect

The 2020 RDA proposed for vitamin B12 ranges between 1.2 and 2.5 mg for non-pregnant women, children, and men (FSSAI 2021).

Correct

The 2020 RDA proposed for vitamin B12 ranges between 1.2 and 2.5 mcg for non-pregnant women, children, and men (FSSAI 2021).

We apologize for this error.

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来源期刊

Birth Defects Research Medicine-Embryology

CiteScore

3.60

自引率

9.50%

发文量

153

期刊介绍： The journal Birth Defects Research publishes original research and reviews in areas related to the etiology of adverse developmental and reproductive outcome. In particular the journal is devoted to the publication of original scientific research that contributes to the understanding of the biology of embryonic development and the prenatal causative factors and mechanisms leading to adverse pregnancy outcomes, namely structural and functional birth defects, pregnancy loss, postnatal functional defects in the human population, and to the identification of prenatal factors and biological mechanisms that reduce these risks. Adverse reproductive and developmental outcomes may have genetic, environmental, nutritional or epigenetic causes. Accordingly, the journal Birth Defects Research takes an integrated, multidisciplinary approach in its organization and publication strategy. The journal Birth Defects Research contains separate sections for clinical and molecular teratology, developmental and reproductive toxicology, and reviews in developmental biology to acknowledge and accommodate the integrative nature of research in this field. Each section has a dedicated editor who is a leader in his/her field and who has full editorial authority in his/her area.