【摘要】SMAD特异性E3泛素蛋白连接酶1通过激活RhoA/ROCK信号通路促进卵巢癌细胞迁移和侵袭。

IF 3.8 3区 医学 Q2 ONCOLOGY
Oncology reports Pub Date : 2025-09-01 Epub Date: 2025-07-04 DOI:10.3892/or.2025.8938
Wei Wang, Han Du, Huiping Liu, Fangfang Hu, Guangzhi Liu
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引用次数: 0

摘要

遵循本文的发表,这是编辑的注意力吸引读者的担心一双Transwell迁移试验的数据面板显示实验图1 B p。671年,和另外两双的数据面板显示入侵和迁移分析实验的结果在图2 a和B p。672年分别包含重叠部分的数据,受影响的面板,这是为了显示不同的结果进行实验,显然都来自同一个原始来源。由于在这篇论文中发现了这些重复的数据面板,《肿瘤学报告》的编辑决定,由于对所呈现的数据缺乏信心,应该从期刊上撤下这篇论文。作者被要求对这些担忧作出解释,但编辑部没有收到答复。对于由此给读者带来的不便,本刊编辑深表歉意。[j] journal of Oncology; 2019;DOI: 10.3892 / or.2018.6836]。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Retracted] SMAD specific E3 ubiquitin protein ligase 1 promotes ovarian cancer cell migration and invasion via the activation of the RhoA/ROCK signaling pathway.

Following the publication of this paper, it was drawn to the Editor's attention by a concerned reader that a pair of the data panels shown for the Transwell migration assay experiments in Fig. 1B on p. 671, and two further pairs of data panels showing the results of invasion and migration assay experiments in Fig. 2A and B on p. 672 respectively, contained overlapping sections of data, such that the affected panels, which were intended to show the results of differently performed experiments, had all apparently been derived from the same original sources. Owing to the identification of these duplicated data panels in this paper, the Editor of Oncology Reports has decided that it should be retracted from the Journal on account of a lack of confidence in the presented data. The authors were asked for an explanation to account for these concerns, but the Editorial Office did not receive a reply. The Editor apologizes to the readership for any inconvenience caused. [Oncology Reports 41: 668‑676, 2019; DOI: 10.3892/or.2018.6836].

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来源期刊
Oncology reports
Oncology reports 医学-肿瘤学
CiteScore
8.50
自引率
2.40%
发文量
187
审稿时长
3 months
期刊介绍: Oncology Reports is a monthly, peer-reviewed journal devoted to the publication of high quality original studies and reviews concerning a broad and comprehensive view of fundamental and applied research in oncology, focusing on carcinogenesis, metastasis and epidemiology.
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