{"title":"对“靶向DQA1*01等位基因家族的HLA-DQ α等位基因52SK的表征”的更正","authors":"","doi":"10.1111/tan.70302","DOIUrl":null,"url":null,"abstract":"<p>M. Devriese, J. H. Lee, T. Meng, et al., “Characterisation of the HLA-DQ Alpha Eplet 52SK Targeting the <i>DQA1*01</i> Alleles Family,” <i>HLA</i> 104, no. 4 (2024): e15700, https://doi.org/10.1111/tan.15700.</p><p>In the article, the French Agence de la Biomédecine (AOR 2022) was missing from the Acknowledgements section.</p><p>The Acknowledgements reads:</p><p>This work was supported by public funding agencies (ANR EPIHLA AAPG2022 to JLT), non-profit organisations (Fondation Victor and Erminia Mescle FRM 2022 to MD) and the commercial sector (Research Services Agreement between One Lambda, Inc. and the French Law 1901 affiliated Association Alloimmunity and Inflammation).</p><p>The Acknowledgements should read:</p><p>This work was supported by public funding agencies (ANR EPIHLA AAPG2022 to JLT), the French Agence de la Biomédecine (AOR 2022), non-profit organisations (Fondation Victor and Erminia Mescle FRM 2022 to MD), and the commercial sector (Research Services Agreement between One Lambda, Inc. and the French Law 1901 affiliated Association Alloimmunity and Inflammation).</p><p>We apologise for this error.</p>","PeriodicalId":13172,"journal":{"name":"HLA","volume":"106 1","pages":""},"PeriodicalIF":5.9000,"publicationDate":"2025-07-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/tan.70302","citationCount":"0","resultStr":"{\"title\":\"Correction to “Characterisation of the HLA-DQ Alpha Eplet 52SK Targeting the DQA1*01 Alleles Family”\",\"authors\":\"\",\"doi\":\"10.1111/tan.70302\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>M. Devriese, J. H. Lee, T. Meng, et al., “Characterisation of the HLA-DQ Alpha Eplet 52SK Targeting the <i>DQA1*01</i> Alleles Family,” <i>HLA</i> 104, no. 4 (2024): e15700, https://doi.org/10.1111/tan.15700.</p><p>In the article, the French Agence de la Biomédecine (AOR 2022) was missing from the Acknowledgements section.</p><p>The Acknowledgements reads:</p><p>This work was supported by public funding agencies (ANR EPIHLA AAPG2022 to JLT), non-profit organisations (Fondation Victor and Erminia Mescle FRM 2022 to MD) and the commercial sector (Research Services Agreement between One Lambda, Inc. and the French Law 1901 affiliated Association Alloimmunity and Inflammation).</p><p>The Acknowledgements should read:</p><p>This work was supported by public funding agencies (ANR EPIHLA AAPG2022 to JLT), the French Agence de la Biomédecine (AOR 2022), non-profit organisations (Fondation Victor and Erminia Mescle FRM 2022 to MD), and the commercial sector (Research Services Agreement between One Lambda, Inc. and the French Law 1901 affiliated Association Alloimmunity and Inflammation).</p><p>We apologise for this error.</p>\",\"PeriodicalId\":13172,\"journal\":{\"name\":\"HLA\",\"volume\":\"106 1\",\"pages\":\"\"},\"PeriodicalIF\":5.9000,\"publicationDate\":\"2025-07-04\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1111/tan.70302\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"HLA\",\"FirstCategoryId\":\"3\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1111/tan.70302\",\"RegionNum\":4,\"RegionCategory\":\"医学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q2\",\"JCRName\":\"CELL BIOLOGY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"HLA","FirstCategoryId":"3","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1111/tan.70302","RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q2","JCRName":"CELL BIOLOGY","Score":null,"Total":0}
引用次数: 0
摘要
李志强，李志强，李志强，等，“HLA- DQA1*01等位基因家族基因多态性的研究”，中华人民大学学报（自然科学版），第4期。4 (2024): e15700, https://doi.org/10.1111/tan.15700.In这篇文章，the French Agence de la biomdecine （AOR 2022）在致谢部分缺失。这项工作得到了公共资助机构（ANR EPIHLA AAPG2022至JLT）、非营利组织（Victor基金会和Erminia Mescle FRM 2022至MD）和商业部门（One Lambda, Inc.与法国法律1901附属协会Alloimmunity and Inflammation之间的研究服务协议）的支持。致谢内容如下：这项工作得到了公共资助机构（ANR EPIHLA AAPG2022至JLT）、法国生物医学化学物质交换（AOR 2022）、非营利组织（Victor基金会和Erminia Mescle FRM 2022至MD）和商业部门（One Lambda, Inc.与法国法律1901附属协会Alloimmunity and Inflammation之间的研究服务协议）的支持。我们为这个错误道歉。
Correction to “Characterisation of the HLA-DQ Alpha Eplet 52SK Targeting the DQA1*01 Alleles Family”
M. Devriese, J. H. Lee, T. Meng, et al., “Characterisation of the HLA-DQ Alpha Eplet 52SK Targeting the DQA1*01 Alleles Family,” HLA 104, no. 4 (2024): e15700, https://doi.org/10.1111/tan.15700.
In the article, the French Agence de la Biomédecine (AOR 2022) was missing from the Acknowledgements section.
The Acknowledgements reads:
This work was supported by public funding agencies (ANR EPIHLA AAPG2022 to JLT), non-profit organisations (Fondation Victor and Erminia Mescle FRM 2022 to MD) and the commercial sector (Research Services Agreement between One Lambda, Inc. and the French Law 1901 affiliated Association Alloimmunity and Inflammation).
The Acknowledgements should read:
This work was supported by public funding agencies (ANR EPIHLA AAPG2022 to JLT), the French Agence de la Biomédecine (AOR 2022), non-profit organisations (Fondation Victor and Erminia Mescle FRM 2022 to MD), and the commercial sector (Research Services Agreement between One Lambda, Inc. and the French Law 1901 affiliated Association Alloimmunity and Inflammation).
期刊介绍：
HLA, the journal, publishes articles on various aspects of immunogenetics. These include the immunogenetics of cell surface antigens, the ontogeny and phylogeny of the immune system, the immunogenetics of cell interactions, the functional aspects of cell surface molecules and their natural ligands, and the role of tissue antigens in immune reactions. Additionally, the journal covers experimental and clinical transplantation, the relationships between normal tissue antigens and tumor-associated antigens, the genetic control of immune response and disease susceptibility, and the biochemistry and molecular biology of alloantigens and leukocyte differentiation. Manuscripts on molecules expressed on lymphoid cells, myeloid cells, platelets, and non-lineage-restricted antigens are welcomed. Lastly, the journal focuses on the immunogenetics of histocompatibility antigens in both humans and experimental animals, including their tissue distribution, regulation, and expression in normal and malignant cells, as well as the use of antigens as markers for disease.
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