严重骨关节炎患者的滑膜液细胞外囊泡促进促分解代谢炎性软骨细胞表型的差异

IF 4.8 2区 生物学 Q1 BIOCHEMISTRY & MOLECULAR BIOLOGY
Biomolecules Pub Date : 2025-06-06 DOI:10.3390/biom15060829
Caitlin Ditchfield, Joshua Price, Edward T Davis, Simon W Jones
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引用次数: 0

摘要

滑膜炎症被认为是骨关节炎(OA)的病理驱动因素,骨关节炎是一种涉及软骨退化和关节疼痛的退行性关节疾病。由于细胞外囊泡(EVs)已成为细胞串扰的关键介质,本研究表征了不同疾病严重程度OA患者的滑液EVs (sevs),并确定了它们对OA关节软骨细胞的功能影响。收集膝关节手术患者的滑液和关节软骨。通过超离心分离sevs,并通过纳米颗粒跟踪分析、ExoView和Luminex分析对蛋白质货物进行表征。根据牛津膝关节评分和EQ5D将患者分为轻度/中度和重度oa组。用sfev处理软骨细胞,使用RNA测序、Luminex和ELISA分析转录组学和分泌组学反应。严重OA患者的sfev更丰富,更小,并表现出四蛋白表达增加。滑液和sevs诱导软骨细胞明显的转录组变化。严重OA患者的SFEVs促进促炎和分解代谢软骨细胞表型,上调CRTAC1、COL6A3、TNC和CXCL5,增加IL-6、MMP1、MMP3和MMP13以及促伤害介质(NGF和P物质)的分泌。这些发现表明sfev可能通过加剧软骨损伤和促进疼痛敏感来促进OA的进展。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Synovial Fluid Extracellular Vesicles from Patients with Severe Osteoarthritis Differentially Promote a Pro-Catabolic, Inflammatory Chondrocyte Phenotype.

Synovial inflammation is recognised as a pathological driver of osteoarthritis (OA), a degenerative joint disease involving cartilage degradation and joint pain. Since extracellular vesicles (EVs) have emerged as key mediators of cellular cross-talk, this study characterised synovial fluid EVs (SFEVs) in OA patients with varying disease severity and determined their functional effects on OA articular chondrocytes. Synovial fluid and articular cartilage were collected from patients undergoing knee surgery. SFEVs were isolated via ultracentrifugation and characterised by nanoparticle tracking analysis, ExoView, and Luminex analysis of protein cargo. Patients were stratified into mild/moderate- and severe-OA groups based on Oxford Knee Score and EQ5D. Chondrocytes were treated with SFEVs, and transcriptomic and secretome responses were analysed using RNA sequencing, Luminex, and ELISA. SFEVs from patients with severe OA were more abundant, smaller and exhibited increased tetraspanin expression. Synovial fluid and SFEVs induced distinct transcriptomic changes in chondrocytes. SFEVs from patients with severe OA promoted a pro-inflammatory and catabolic chondrocyte phenotype, with upregulation of CRTAC1, COL6A3, TNC, and CXCL5, greater secretion of IL-6, MMP1, MMP3 and MMP13, and pro-nociceptive mediators (NGF and Substance P). These findings suggest that SFEVs may contribute to OA progression by exacerbating cartilage damage and promoting pain sensitisation.

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来源期刊
Biomolecules
Biomolecules Biochemistry, Genetics and Molecular Biology-Molecular Biology
CiteScore
9.40
自引率
3.60%
发文量
1640
审稿时长
18.28 days
期刊介绍: Biomolecules (ISSN 2218-273X) is an international, peer-reviewed open access journal focusing on biogenic substances and their biological functions, structures, interactions with other molecules, and their microenvironment as well as biological systems. Biomolecules publishes reviews, regular research papers and short communications.  Our aim is to encourage scientists to publish their experimental and theoretical results in as much detail as possible. There is no restriction on the length of the papers. The full experimental details must be provided so that the results can be reproduced.
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