研究miRNA-137-3p/383-5p/PGC-1α信号通路对心肌肥厚的治疗潜力。

IF 2.4 3区 医学 Q2 CARDIAC & CARDIOVASCULAR SYSTEMS
Khadam Hussain, Somia Khurram, Muhammad Yousaf, Ayesha Ishtiaq, Iram Mushtaq, Tahir Ali, Iram Murtaza
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引用次数: 0

摘要

心肌细胞的病理生长称为心肌肥厚(CH)。mirna的差异表达对心脏肥厚具有巨大的治疗潜力。本研究旨在通过调控PGC-1α信号通路,评估miRNA-137-3p/383-5p在心肌肥厚中的治疗潜力。miR-137-3p和miR-383-5p等促增生性mirna的沉默导致其共同靶基因PGC-1α在增生性细胞中的恢复。有趣的是,这项体内研究的结果显示了这些抗戈米的心脏保护作用。此外,PGC-1α相关的信号事件,如脂肪酸氧化(Cpt1a, Cpt1b),线粒体膜电位(MMP),线粒体活性氧(mtROS),氧化磷酸化(Ndufa6, Atp5me),凋亡(Bcl-2, BAX),抗氧化剂(SOD, GSH, CAT),线粒体动力学(Mfn-2, Drp-1)在miRNA拮抗剂处理组中显著恢复。最后,本研究揭示了miR-137-3p和miR-383-5p的药理抑制可能通过调节PGC-1α信号通路来挽救心脏肥厚。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Investigating the Therapeutic Potential of miRNA-137-3p/383-5p/PGC-1α Signalling Nexus Against Cardiac Hypertrophy.

Pathological growth of cardiomyocytes known as cardiac hypertrophy (CH). Differential expressions of miRNAs have an immense therapeutic potential against cardiac hypertrophy. The current study aim is to evaluate the therapeutic potential of miRNA-137-3p/383-5p in cardiac hypertrophy by regulation of PGC-1α signaling nexus. Silencing of pro-hypertrophic miRNAs e.g. miR-137-3p and miR-383-5p leads to the restoration of their common target gene PGC-1α in hypertrophic cells. Interestingly, the results of this invivo study showed the cardioprotective effects of these antagomirs. Moreover, PGC-1α associated signaling events e.g. fatty acid oxidation (Cpt1a, Cpt1b), mitochondria membrane potential (MMP), mitochondrial reactive oxygen species (mtROS), oxidative phosphorylation (Ndufa6, Atp5me), apoptosis (Bcl-2, BAX), antioxidants (SOD, GSH, CAT), mitochondrial dynamic (Mfn-2, Drp-1) were significantly restored in the treated groups of miRNA antagomirs. Conclusively, this study uncovers that the pharmacological inhibition of miR-137-3p and miR-383-5p have a potential to rescue from the cardiac hypertrophy by regulation of PGC-1α signaling nexus.

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来源期刊
Journal of Cardiovascular Translational Research
Journal of Cardiovascular Translational Research CARDIAC & CARDIOVASCULAR SYSTEMS-MEDICINE, RESEARCH & EXPERIMENTAL
CiteScore
6.10
自引率
2.90%
发文量
148
审稿时长
6-12 weeks
期刊介绍: Journal of Cardiovascular Translational Research (JCTR) is a premier journal in cardiovascular translational research. JCTR is the journal of choice for authors seeking the broadest audience for emerging technologies, therapies and diagnostics, pre-clinical research, and first-in-man clinical trials. JCTR''s intent is to provide a forum for critical evaluation of the novel cardiovascular science, to showcase important and clinically relevant aspects of the new research, as well as to discuss the impediments that may need to be overcome during the translation to patient care.
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