Yun Jae Cha, Young-Kwang Kim, Yun Ji Lim, Chan Ho Kim, Se Eun Park, Yun Pyo Kang, Min-Kyoo Shin
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Notably, this lipid profile was also observed in microglia cells overexpressing the Swedish mutant form of Aβ precursor protein (APPswe). Given the critical role of BMP in lysosomal lipid and membrane degradation, and the observed enhancement of GM3 accumulation under lysosomal inhibition in APPswe-transfected microglial cells, these findings suggest that Aβ-mediated microglial lysosomal dysfunction may contribute to AD progression. 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引用次数: 0
摘要
5xFAD小鼠模型是研究阿尔茨海默病(AD),特别是淀粉样蛋白- β (a β)诱导的AD病理的有价值的实验系统。在这项研究中,我们探索了5xFAD小鼠皮质和海马内脂质组的时间、区域和性别特异性改变。我们的研究结果显示,随着大脑皮层和海马中Aβ病理的进展,脂质改变变得更加明显。这些脂质变化在雌性小鼠中也更为显著,表现出比雄性小鼠更严重的Aβ病理。通过脂质网络分析,我们在两个脑区发现了ad特异性脂质共表达网络模块,以BMP和GM3等溶酶体脂质的富集为标志。值得注意的是,在Aβ前体蛋白瑞典突变体(APPswe)过表达的小胶质细胞中也观察到这种脂质谱。考虑到BMP在溶酶体脂质和膜降解中的关键作用,以及在appswee转染的小胶质细胞中观察到的溶酶体抑制下GM3积累的增强,这些研究结果表明,a β介导的小胶质溶酶体功能障碍可能有助于AD的进展。总的来说,我们发现了先前未被认识到的a β在溶酶体脂质代谢失调中的作用,并强调了脂质组学和网络分析作为阐明疾病机制的补充方法的实用性。
Lipidomic Network Analysis Reveals Amyloid-β-Induced Lysosomal Lipid Accumulation in the Cortex and Hippocampus of 5xFAD Mice.
The 5xFAD mouse model serves as a valuable experimental system for investigating Alzheimer's disease (AD), specifically amyloid-beta (Aβ)-induced AD pathology. In this study, we explored temporal, regional, and sex-specific alternations in the lipidome within the cortex and hippocampus of 5xFAD mice. Our results revealed that lipid alternations become more pronounced with the progression of Aβ pathology in the cerebral cortex and hippocampus. These lipid changes were also more significant in the female mice, which exhibited more severe Aβ pathology than male mice. Through lipid network analysis, we identified AD-specific lipid coexpression network modules in both brain regions, marked by enriched lysosomal lipids such as BMP and GM3. Notably, this lipid profile was also observed in microglia cells overexpressing the Swedish mutant form of Aβ precursor protein (APPswe). Given the critical role of BMP in lysosomal lipid and membrane degradation, and the observed enhancement of GM3 accumulation under lysosomal inhibition in APPswe-transfected microglial cells, these findings suggest that Aβ-mediated microglial lysosomal dysfunction may contribute to AD progression. Overall, we discovered a previously unrecognized role of Aβ in dysregulating lysosomal lipid metabolism and highlighted the utility of lipidomics and network analysis as complementary approaches for elucidating disease mechanisms.
期刊介绍:
Journal of Proteome Research publishes content encompassing all aspects of global protein analysis and function, including the dynamic aspects of genomics, spatio-temporal proteomics, metabonomics and metabolomics, clinical and agricultural proteomics, as well as advances in methodology including bioinformatics. The theme and emphasis is on a multidisciplinary approach to the life sciences through the synergy between the different types of "omics".