{"title":"对“环状RNA NFIX通过调节miR-214-3p/TRIAP1轴在非小细胞肺癌中起致癌基因作用”的更正","authors":"","doi":"10.1111/crj.70096","DOIUrl":null,"url":null,"abstract":"<p>\n <span>G. Liu</span>, <span>H. Shi</span>, <span>H. Zheng</span>, <span>W. Kong</span>, <span>X. Cheng</span>, <span>L. Deng</span>, “ <span>Circular RNA NFIX Functions as an Oncogene in Non-Small Cell Lung Cancer by Modulating the miR-214-3p/TRIAP1 Axis</span>,” <i>Clinical Respiratory Journal</i> <span>18</span>, no. <span>8</span> (<span>2024</span>). https://doi.org/10.1111/crj.13801.\n </p><p>Figures 4 and 7 are incorrect. Below are the correct figures.</p><p>FIGURE 4 | Effect of circRNA NFIX interference in NSCLC by targeting miRNA-214-3p. A549 cells were transfected with control-siRNA, circRNA NFIX-siRNA, circRNA NFIX-siRNA + inhibitor control, or circRNA NFIX-siRNA + miR-214-3p inhibitor for 48 h. (A) Cell proliferation was counted by MTT assay. (B and C) The apoptosis ratio of cancer cells was detected by flow cytometry. (D and E) The level of cleaved-caspase3 was detected by western blot assay, and cleaved-caspase3/caspase3 ratio was calculated. ** indicates <i>p</i> < 0.01 versus control-siRNA, ## indicates <i>p</i> < 0.01 circRNA NFIX-siRNA + inhibitor control. Data are exhibited as average ± SD of triple single experiments.</p><p>FIGURE 7 | Overexpression of miRNA-214-3p suppressed cell growth and promoted cell apoptosis via TRIAP1 in NSCLC cells. A549 cells were transfected with mimic control, miRNA-214-3p mimic, miRNA-214-3p mimic + control-plasmid, or miRNA-214-3p mimic + TRIAP1-plasmid for 48 h. (A) Cell proliferation was counted by MTT assay. (B and C) The apoptosis of A549 cells was analyzed by flow cytometry. (D and E) The level of cleaved-caspase3 was detected by western blot assay, and the ratio of cleaved-caspase3/caspase3 was determined. ** indicates <i>p</i> < 0.01 versus mimic control, ## indicates <i>p</i> < 0.01 versus miR-214-3p mimic + control-plasmid. Data are exhibited as average ± SD of triple single experiments.</p><p>We apologize for these errors.</p>","PeriodicalId":55247,"journal":{"name":"Clinical Respiratory Journal","volume":"19 6","pages":""},"PeriodicalIF":1.9000,"publicationDate":"2025-06-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/crj.70096","citationCount":"0","resultStr":"{\"title\":\"Correction to “Circular RNA NFIX Functions as an Oncogene in Non-Small Cell Lung Cancer by Modulating the miR-214-3p/TRIAP1 Axis”\",\"authors\":\"\",\"doi\":\"10.1111/crj.70096\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>\\n <span>G. Liu</span>, <span>H. Shi</span>, <span>H. Zheng</span>, <span>W. Kong</span>, <span>X. Cheng</span>, <span>L. Deng</span>, “ <span>Circular RNA NFIX Functions as an Oncogene in Non-Small Cell Lung Cancer by Modulating the miR-214-3p/TRIAP1 Axis</span>,” <i>Clinical Respiratory Journal</i> <span>18</span>, no. <span>8</span> (<span>2024</span>). https://doi.org/10.1111/crj.13801.\\n </p><p>Figures 4 and 7 are incorrect. Below are the correct figures.</p><p>FIGURE 4 | Effect of circRNA NFIX interference in NSCLC by targeting miRNA-214-3p. A549 cells were transfected with control-siRNA, circRNA NFIX-siRNA, circRNA NFIX-siRNA + inhibitor control, or circRNA NFIX-siRNA + miR-214-3p inhibitor for 48 h. (A) Cell proliferation was counted by MTT assay. (B and C) The apoptosis ratio of cancer cells was detected by flow cytometry. (D and E) The level of cleaved-caspase3 was detected by western blot assay, and cleaved-caspase3/caspase3 ratio was calculated. ** indicates <i>p</i> < 0.01 versus control-siRNA, ## indicates <i>p</i> < 0.01 circRNA NFIX-siRNA + inhibitor control. Data are exhibited as average ± SD of triple single experiments.</p><p>FIGURE 7 | Overexpression of miRNA-214-3p suppressed cell growth and promoted cell apoptosis via TRIAP1 in NSCLC cells. A549 cells were transfected with mimic control, miRNA-214-3p mimic, miRNA-214-3p mimic + control-plasmid, or miRNA-214-3p mimic + TRIAP1-plasmid for 48 h. (A) Cell proliferation was counted by MTT assay. (B and C) The apoptosis of A549 cells was analyzed by flow cytometry. (D and E) The level of cleaved-caspase3 was detected by western blot assay, and the ratio of cleaved-caspase3/caspase3 was determined. ** indicates <i>p</i> < 0.01 versus mimic control, ## indicates <i>p</i> < 0.01 versus miR-214-3p mimic + control-plasmid. 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Correction to “Circular RNA NFIX Functions as an Oncogene in Non-Small Cell Lung Cancer by Modulating the miR-214-3p/TRIAP1 Axis”
G. Liu, H. Shi, H. Zheng, W. Kong, X. Cheng, L. Deng, “ Circular RNA NFIX Functions as an Oncogene in Non-Small Cell Lung Cancer by Modulating the miR-214-3p/TRIAP1 Axis,” Clinical Respiratory Journal18, no. 8 (2024). https://doi.org/10.1111/crj.13801.
Figures 4 and 7 are incorrect. Below are the correct figures.
FIGURE 4 | Effect of circRNA NFIX interference in NSCLC by targeting miRNA-214-3p. A549 cells were transfected with control-siRNA, circRNA NFIX-siRNA, circRNA NFIX-siRNA + inhibitor control, or circRNA NFIX-siRNA + miR-214-3p inhibitor for 48 h. (A) Cell proliferation was counted by MTT assay. (B and C) The apoptosis ratio of cancer cells was detected by flow cytometry. (D and E) The level of cleaved-caspase3 was detected by western blot assay, and cleaved-caspase3/caspase3 ratio was calculated. ** indicates p < 0.01 versus control-siRNA, ## indicates p < 0.01 circRNA NFIX-siRNA + inhibitor control. Data are exhibited as average ± SD of triple single experiments.
FIGURE 7 | Overexpression of miRNA-214-3p suppressed cell growth and promoted cell apoptosis via TRIAP1 in NSCLC cells. A549 cells were transfected with mimic control, miRNA-214-3p mimic, miRNA-214-3p mimic + control-plasmid, or miRNA-214-3p mimic + TRIAP1-plasmid for 48 h. (A) Cell proliferation was counted by MTT assay. (B and C) The apoptosis of A549 cells was analyzed by flow cytometry. (D and E) The level of cleaved-caspase3 was detected by western blot assay, and the ratio of cleaved-caspase3/caspase3 was determined. ** indicates p < 0.01 versus mimic control, ## indicates p < 0.01 versus miR-214-3p mimic + control-plasmid. Data are exhibited as average ± SD of triple single experiments.
期刊介绍：
Overview
Effective with the 2016 volume, this journal will be published in an online-only format.
Aims and Scope
The Clinical Respiratory Journal (CRJ) provides a forum for clinical research in all areas of respiratory medicine from clinical lung disease to basic research relevant to the clinic.
We publish original research, review articles, case studies, editorials and book reviews in all areas of clinical lung disease including:
Asthma
Allergy
COPD
Non-invasive ventilation
Sleep related breathing disorders
Interstitial lung diseases
Lung cancer
Clinical genetics
Rhinitis
Airway and lung infection
Epidemiology
Pediatrics
CRJ provides a fast-track service for selected Phase II and Phase III trial studies.
Keywords
Clinical Respiratory Journal, respiratory, pulmonary, medicine, clinical, lung disease,
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