Pnky通过激活Wnt/β-Catenin信号通路调节神经干细胞的增殖和分化。

IF 1.6 4区 生物学 Q4 BIOCHEMISTRY & MOLECULAR BIOLOGY
Organogenesis Pub Date : 2025-12-01 Epub Date: 2025-06-16 DOI:10.1080/15476278.2025.2519641
Haidong Wu, Jing Huang, Xiaojing Li, Yali Song, Xuxiang Chen, Yajie Guo
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引用次数: 0

摘要

神经干细胞(NSC)具有多能性和自我更新的基本特性,使其成为治疗阿尔茨海默病(AD)、帕金森病(PD)和脊髓损伤等神经系统疾病的有希望的候选者。虽然之前的研究已经确定了长链非编码RNA (lncRNAs) Pnky是通过RNA剪接介导NSC向神经元分化的调节剂,但其通过Wnt/β-catenin通路在NSC分化和增殖中的作用尚不清楚。在本研究中,我们研究了Pnky通过影响Wnt/β-catenin通路促进NSC向神经元分化的机制。通过cck8检测、western blot分析和定量聚合酶链反应(quantitative polymerase chain reaction, qPCR),我们发现Pnky基因敲低可显著增强NSC的增殖,促进其向神经元分化。此外,Pnky敲低通过激活β-catenin信号通路,导致神经干细胞标记物Nestin下调,神经元标记物β3-Tubulin上调。相反,抑制β-catenin通路会阻碍NSC的分化和增殖。这些发现表明,靶向pnky介导的Wnt/β-catenin通路可能为中枢神经系统疾病的治疗、诊断和药物开发提供新的策略。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Pnky Modulates Neural Stem Cell Proliferation and Differentiation Through Activation of Wnt/β-Catenin Signaling Pathway.

Neural stem cell (NSC) possess the essential properties of pluripotency and self-renewal, making them promising candidates for the treatment of neurological disorders such as Alzheimer's disease (AD), Parkinson's disease (PD), and spinal cord injuries. While previous studies have identified the long non-coding RNAs (lncRNAs) Pnky as a regulator of NSC differentiation into neurons via RNA splicing, its role in NSC differentiation and proliferation through the Wnt/β-catenin pathway remains unclear. In this study, we investigated the mechanism by which Pnky influences the Wnt/β-catenin pathway to promote NSC differentiation into neurons. Using cck8 assays, western blot analysis, and quantitative polymerase chain reaction (qPCR), we found that Pnky knockdown significantly enhanced NSC proliferation and promoted their differentiation into neurons. Additionally, Pnky knockdown resulted in the downregulation of the neural stem cell marker Nestin and upregulation of the neuronal marker β3-Tubulin, through activation of the β-catenin signaling pathway. Conversely, inhibiting the β-catenin pathway hindered both NSC differentiation and proliferation. These findings suggest that targeting the Pnky-mediated Wnt/β-catenin pathway may offer novel strategies for the treatment, diagnosis, and drug development of central nervous system diseases.

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来源期刊
Organogenesis
Organogenesis BIOCHEMISTRY & MOLECULAR BIOLOGY-DEVELOPMENTAL BIOLOGY
CiteScore
4.10
自引率
4.30%
发文量
6
审稿时长
>12 weeks
期刊介绍: Organogenesis is a peer-reviewed journal, available in print and online, that publishes significant advances on all aspects of organ development. The journal covers organogenesis in all multi-cellular organisms and also includes research into tissue engineering, artificial organs and organ substitutes. The overriding criteria for publication in Organogenesis are originality, scientific merit and general interest. The audience of the journal consists primarily of researchers and advanced students of anatomy, developmental biology and tissue engineering. The emphasis of the journal is on experimental papers (full-length and brief communications), but it will also publish reviews, hypotheses and commentaries. The Editors encourage the submission of addenda, which are essentially auto-commentaries on significant research recently published elsewhere with additional insights, new interpretations or speculations on a relevant topic. If you have interesting data or an original hypothesis about organ development or artificial organs, please send a pre-submission inquiry to the Editor-in-Chief. You will normally receive a reply within days. All manuscripts will be subjected to peer review, and accepted manuscripts will be posted to the electronic site of the journal immediately and will appear in print at the earliest opportunity thereafter.
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