血清素神经调节指导视神经再生。

IF 3.7 2区 生物学 Q1 DEVELOPMENTAL BIOLOGY
Development Pub Date : 2025-06-16 DOI:10.1242/dev.204334
Kristian Saied-Santiago, Melissa Baxter, Jaffna Mathiaparanam, Michael Granato
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引用次数: 0

摘要

视神经(ON)再生在哺乳动物系统是有限的阴影优势的抑制因素。这严重阻碍了对促再生途径的识别。在这里,我们利用斑马鱼幼体的再生能力来确定促进ON再生的途径。从小分子筛选,我们确定了5-羟色胺(5-HT)信号调节抑制ON再生。我们发现几个5-羟色胺1型受体基因在再生过程中在RGC神经元中表达,抑制5-羟色胺1受体或5-羟色胺途径的成分选择性地阻碍ON再生。我们发现5-HT1受体信号在ON发育过程中是不可缺少的,但对于再生轴突从损伤部位出现至关重要。一旦ON轴突越过交叉,阻断5-HT受体不会抑制再生,这表明在ON再生过程中,5-HT受体的早期信号传导具有选择性作用。最后,我们发现激动剂介导的5-HT1受体激活导致轴突再生增强和异位。综上所述,我们的结果为血清素依赖性神经调节指导体内ON再生的机制提供了证据。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Serotonin neuromodulation directs optic nerve regeneration.

Optic nerve (ON) regeneration in mammalian systems is limited by an overshadowing dominance of inhibitory factors. This has severely hampered the identification of pro-regenerative pathways. Here, we take advantage of the regenerative capacity of larval zebrafish to identify pathways that promote ON regeneration. From a small molecule screen, we identified modulators of serotonin (5-HT) signaling that inhibit ON regeneration. We find several serotonin type-1 receptor genes are expressed in RGC neurons during regeneration and that inhibiting 5-HT1 receptors or components of the 5-HT pathway selectively impedes ON regeneration. We show that 5-HT1 receptor signaling is dispensable during ON development yet is critical for regenerating axons to emerge from the injury site. Blocking 5-HT receptors once ON axons have crossed the chiasm does not inhibit regeneration, suggesting a selective role for 5-HT receptor signaling early during ON regeneration. Finally, we show that agonist-mediated activation of 5-HT1 receptors leads to enhanced and ectopic axonal regrowth. Combined, our results provide evidence for mechanisms through which serotonin-dependent neuromodulation directs ON regeneration in vivo.

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来源期刊
Development
Development 生物-发育生物学
CiteScore
6.70
自引率
4.30%
发文量
433
审稿时长
3 months
期刊介绍: Development’s scope covers all aspects of plant and animal development, including stem cell biology and regeneration. The single most important criterion for acceptance in Development is scientific excellence. Research papers (articles and reports) should therefore pose and test a significant hypothesis or address a significant question, and should provide novel perspectives that advance our understanding of development. We also encourage submission of papers that use computational methods or mathematical models to obtain significant new insights into developmental biology topics. Manuscripts that are descriptive in nature will be considered only when they lay important groundwork for a field and/or provide novel resources for understanding developmental processes of broad interest to the community. Development includes a Techniques and Resources section for the publication of new methods, datasets, and other types of resources. Papers describing new techniques should include a proof-of-principle demonstration that the technique is valuable to the developmental biology community; they need not include in-depth follow-up analysis. The technique must be described in sufficient detail to be easily replicated by other investigators. Development will also consider protocol-type papers of exceptional interest to the community. We welcome submission of Resource papers, for example those reporting new databases, systems-level datasets, or genetic resources of major value to the developmental biology community. For all papers, the data or resource described must be made available to the community with minimal restrictions upon publication. To aid navigability, Development has dedicated sections of the journal to stem cells & regeneration and to human development. The criteria for acceptance into these sections is identical to those outlined above. Authors and editors are encouraged to nominate appropriate manuscripts for inclusion in one of these sections.
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