新型脂噬诱导剂作为脂代谢紊乱的潜在治疗药物。

IF 3.5 2区 生物学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY
Rachel Njeim, Bassel Awada, Haley Donow, Haley Gye, Cole Foster, Colin Kelly, Judith Molina, Sandra Merscher, Marcello Giulianotti, Alessia Fornoni, Hassan Al-Ali
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引用次数: 0

摘要

脂质稳态失调与广泛的病理相关,包括神经、代谢、心血管、肿瘤和肾脏疾病。我们之前的研究表明,足细胞中的脂滴(LD)积累有助于糖尿病肾病(DKD)的进展,减少脂滴可保持足细胞功能并防止蛋白尿。在这里,我们试图确定治疗病理性LD积累的化合物。我们开发了一种使用人类足细胞的表型测定方法,并将其用于筛选包含超过4500万个独特小分子的组合文库。这导致鉴定化合物系列,有效地减少应激足细胞的LD积累。机制研究表明,这些化合物可激活脂噬,减少LD积累,并从细胞死亡中拯救足细胞。相比之下,已知的诱导一般自噬的化合物无法模仿这些作用,这表明一种新的脂噬特异性作用机制(MoA),这是由无偏倚表型分析证实的。这种治疗策略的一个优点是它不仅可以阻止病理性脂质积累的进展,而且可以逆转它。这些化合物将作为发现新的药物靶点和治疗moa的工具,用于治疗DKD和其他病因相似的疾病。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Novel Lipophagy Inducers as Potential Therapeutics for Lipid Metabolism Disorders.

Dysregulation of lipid homeostasis is associated with a wide range of pathologies encompassing neurological, metabolic, cardiovascular, oncological, and renal disorders. We previously showed that lipid droplet (LD) accumulation in podocytes contributes to the progression of diabetic kidney disease (DKD) and reducing LDs preserves podocyte function and prevents albuminuria. Here, we sought to identify compounds that treat pathological LD accumulation. We developed a phenotypic assay using human podocytes and deployed it to screen a combinatorial library comprising over 45 million unique small molecules. This led to the identification of a compound series that effectively reduces LD accumulation in stressed podocytes. Mechanistic studies revealed that these compounds activate lipophagy, reduce LD accumulation, and rescue podocytes from cell death. In contrast, compounds known to induce general autophagy failed to mimic these effects, indicating a novel lipophagy-specific mechanism of action (MoA), which was confirmed by unbiased phenotypic profiling. An advantage of this therapeutic strategy is its potential to not only halt the progression of pathological lipid accumulation but also reverse it. These compounds will serve as tools for uncovering novel drug targets and therapeutic MoAs for treating DKD and other diseases with similar etiologies.

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来源期刊
ACS Chemical Biology
ACS Chemical Biology 生物-生化与分子生物学
CiteScore
7.50
自引率
5.00%
发文量
353
审稿时长
3.3 months
期刊介绍: ACS Chemical Biology provides an international forum for the rapid communication of research that broadly embraces the interface between chemistry and biology. The journal also serves as a forum to facilitate the communication between biologists and chemists that will translate into new research opportunities and discoveries. Results will be published in which molecular reasoning has been used to probe questions through in vitro investigations, cell biological methods, or organismic studies. We welcome mechanistic studies on proteins, nucleic acids, sugars, lipids, and nonbiological polymers. The journal serves a large scientific community, exploring cellular function from both chemical and biological perspectives. It is understood that submitted work is based upon original results and has not been published previously.
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