调节性T细胞耗竭促进髓细胞活化和胶质母细胞瘤对抗pd1和肿瘤靶向抗体的反应

IF 26.3 1区医学 Q1 IMMUNOLOGY

Immunity Pub Date : 2025-05-29 DOI:10.1016/j.immuni.2025.05.019

Felipe Galvez-Cancino, Mariela Navarrete, Gordon Beattie, Simone Puccio, Enrique Conde-Gallastegi, Kane Foster, Yasmin Morris, Teerapon Sahwangarrom, Despoina Karagianni, Jiali Liu, Alvin J.X. Lee, Dimitrios A. Garyfallos, Alexander P. Simpson, Gerasimos-Theodoros Mastrokalos, Francesco Nannini, Cristobal Costoya, Varshaa Anantharam, Beatrice Claudia Cianciotti, Leanne Bradley, Claudia Garcia-Diaz, Sergio A. Quezada

引用次数: 0

摘要

(免疫58,1236-1253.e1-e8；2025年5月13日)

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Regulatory T cell depletion promotes myeloid cell activation and glioblastoma response to anti-PD1 and tumor-targeting antibodies

(Immunity 58, 1236–1253.e1–e8; May 13, 2025)

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来源期刊

Immunity 医学-免疫学

CiteScore

49.40

自引率

2.20%

发文量

205

审稿时长

6 months

期刊介绍： Immunity is a publication that focuses on publishing significant advancements in research related to immunology. We encourage the submission of studies that offer groundbreaking immunological discoveries, whether at the molecular, cellular, or whole organism level. Topics of interest encompass a wide range, such as cancer, infectious diseases, neuroimmunology, autoimmune diseases, allergies, mucosal immunity, metabolic diseases, and homeostasis.