怀孕期间因生理性垂体增生导致的视力丧失。

IF 0.8 Q4 OBSTETRICS & GYNECOLOGY
Andrew Peel, Stephanie Teasdale, Adam Morton
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引用次数: 0

摘要

生理性垂体增生是由腺垂体细胞数量增加引起的,其特征是垂体弥漫性等致密增大,超出了与年龄相符的大小。它最常见于经历生殖变化(青春期、怀孕、更年期)的女性。我们报告一例垂体增生(垂体高度14mm)在33岁的女性在妊娠34周表现为视力改变。计算机视野识别右侧颞上视野丧失,当前视网膜神经纤维层评估提示压缩性视神经病变。进行了连续的视觉评估,由于进展,在妊娠38周时进行了剖腹产。未使用卡麦角林,分娩一个月后,视觉表现消失,垂体高度降至11mm。关于垂体增生引起的压迫性神经病变的治疗资料有限,与妊娠无关。本病例强调了妊娠期肥厚的暂时性,保守干预的视觉效果良好。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Visual loss during pregnancy due to physiological pituitary hyperplasia.

Physiological pituitary hyperplasia is due to adenohypophyseal cell number increase and is characterised by diffuse isodense pituitary enlargement beyond age-appropriate sizes. It most commonly occurs in females undergoing reproductive changes (puberty, pregnancy, menopause). We report a case of pituitary hyperplasia (pituitary height 14 mm) in a 33-year-old female presenting as vision changes at 34 weeks 'gestation. Computer visual fields identified right sided superotemporal visual field loss with current retinal nerve fibre layer assessment suggestive of compressive optic neuropathy. Serial visual assessment was performed, and due to progression, a caesarean section was performed at 38 weeks' gestation. Cabergoline was not utilised, and one month following delivery, visual findings had resolved, and pituitary height had reduced to 11 mm. There is limited data regarding management for pituitary hyperplasia causing compressive neuropathy irrespective of pregnancy. This case highlights the temporary nature of hypertrophy during pregnancy and positive visual outcome with conservative interventions.

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来源期刊
Obstetric Medicine
Obstetric Medicine OBSTETRICS & GYNECOLOGY-
CiteScore
1.90
自引率
0.00%
发文量
60
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