2型血管性血友病VWF变异的景观和谱:来自德国患者队列的见解

IF 4.3 2区 医学 Q1 HEMATOLOGY
Hamideh Yadegari, Susan Halimeh, Alexander Krahforst, Anna Pavlova, Behnaz Pezeshkpoor, Jens Müller, Bernd Pötzsch, Arijit Biswas, Natascha Marquardt, Ute Scholz, Heinrich Richter, Heiner Trobisch, Karin Liebscher, Martin Olivieri, Karolin Trautmann-Grill, Oliver Tiebel, Ralf Knöfler, Johannes Oldenburg
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引用次数: 0

摘要

血管性血友病(VWD) 2型是由血管性血友病因子(VWF)变异引起的,这种变异破坏了其基本的止血功能。根据ISTH指南,根据受影响的VWF角色,将其分类为2A、2B、2M和2N类型。目的:这项基于人群的研究旨在揭示2型VWD的基因型和实验室表型,为潜在的遗传学和基因型-表型关联提供见解。患者/方法:我们的队列包括来自196个家庭的247名患者。通过多种VWF表型分析和遗传分析,包括DNA测序、拷贝数变异评估和生物信息学评估,对患者进行了表征。结果:86例患者(IPs, 44%)被诊断为2A型,是最常见的亚型。此外,27名ip(14%)被诊断为2N型,24名ip(12%)被诊断为2B型,17名ip(9%)被诊断为2M型,42名ip被归类为U型VWD,携带VWD相关变异,但不能分配到特定亚型。196个个体中有187个(95%)检测到VWF变异。共鉴定出222个VWF变异:187个错义等位基因(84%),22个无效等位基因(10%),5个调节等位基因(2%),6个基因转换等位基因(3%),2个沉默等位基因(1%)。许多变体在我们的队列中反复出现,导致114种不同的变体。其中,45例(39%)是新颖的。结论:我们的数据扩展了VWF疾病相关变异的谱,包括许多新发现的变异。这为准确诊断和个性化治疗提供了有价值的见解。此外,2型患者显著的遗传异质性突出了亚分类的挑战。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Landscape and Spectrum of VWF Variants in Type 2 Von Willebrand Disease: Insights from a German Patient Cohort.

von Willebrand disease (VWD) type 2 arises from variants in von Willebrand factor (VWF) that disrupt its essential hemostatic functions. As per ISTH guidelines, it is classified as type 2A, 2B, 2M, and 2N based on the affected VWF roles.This population-based study aims to uncover the genotype and laboratory phenotypes in type 2 VWD, providing insights into underlying genetics and genotype-phenotype associations.Our cohort included 247 patients from 196 families. Patients were characterized through multiple VWF phenotypic assays and genetic analyses, including DNA sequencing, copy number variation evaluations, and bioinformatic assessments.A total of 86 index patients (IPs, 44%) were diagnosed with type 2A, the most prevalent subtype. Additionally, 27 IPs (14%) were diagnosed with type 2N, 24 IPs (12%) with type 2B, 17 IPs (9%) with type 2M, and 42 IPs categorized as type U VWD carried VWD-associated variants but could not be assigned to a specific subtype. VWF variants were detected in 187 out of 196 (95%) individuals. A total of 222 VWF variants were identified: 187 missense (84%), 22 null alleles (10%), 5 regulatory (2%), 6 gene conversions (3%), and 2 silent variants (1%). Many variants were recurrent in our cohort, resulting in 114 distinct variants. Of these, 45 (39%) were novel.Our data expands the spectrum of disease-associated variants in VWF, including many newly identified variants. This provides valuable insights for accurate diagnosis and personalized treatment. Additionally, the significant genetic heterogeneity among type 2 patients highlights the challenges in sub-classification.

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来源期刊
Thrombosis and haemostasis
Thrombosis and haemostasis 医学-外周血管病
CiteScore
11.90
自引率
9.00%
发文量
140
审稿时长
1 months
期刊介绍: Thrombosis and Haemostasis publishes reports on basic, translational and clinical research dedicated to novel results and highest quality in any area of thrombosis and haemostasis, vascular biology and medicine, inflammation and infection, platelet and leukocyte biology, from genetic, molecular & cellular studies, diagnostic, therapeutic & preventative studies to high-level translational and clinical research. The journal provides position and guideline papers, state-of-the-art papers, expert analysis and commentaries, and dedicated theme issues covering recent developments and key topics in the field.
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